Agency
National Institute of Health (NIH)
Institute
National Institute on Alcohol Abuse and Alcoholism (NIAAA)
Type
Research Scientist Development Award - Research (K02)
Project #
5K02AA000173-03
Application #
2042847
Study Section
Biochemistry, Physiology and Medicine Subcommittee (ALCB)
Project Start
1994-02-01
Project End
1999-01-31
Budget Start
1996-02-01
Budget End
1997-01-31
Support Year
3
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Johns Hopkins University
Department
Internal Medicine/Medicine
Type
Schools of Medicine
DUNS #
045911138
City
Baltimore
State
MD
Country
United States
Zip Code
21218
Rashid, A; Wu, T C; Huang, C C et al. (1999) Mitochondrial proteins that regulate apoptosis and necrosis are induced in mouse fatty liver. Hepatology 29:1131-8
Loffreda, S; Rai, R; Yang, S Q et al. (1997) Bile ducts and portal and central veins are major producers of tumor necrosis factor alpha in regenerating rat liver. Gastroenterology 112:2089-98
Rai, R M; Loffreda, S; Karp, C L et al. (1997) Kupffer cell depletion abolishes induction of interleukin-10 and permits sustained overexpression of tumor necrosis factor alpha messenger RNA in the regenerating rat liver. Hepatology 25:889-95
Diehl, A M; Johns, D C; Yang, S et al. (1996) Adenovirus-mediated transfer of CCAAT/enhancer-binding protein-alpha identifies a dominant antiproliferative role for this isoform in hepatocytes. J Biol Chem 271:7343-50
Westwick, J K; Fleckenstein, J; Yin, M et al. (1996) Differential regulation of hepatocyte DNA synthesis by cAMP in vitro in vivo. Am J Physiol 271:G780-90
Zeldin, G; Yang, S Q; Yin, M et al. (1996) Alcohol and cytokine-inducible transcription factors. Alcohol Clin Exp Res 20:1639-45
Diehl, A M; Yin, M; Fleckenstein, J et al. (1994) Tumor necrosis factor-alpha induces c-jun during the regenerative response to liver injury. Am J Physiol 267:G552-61