HIV-1 infection is characterized by progressive immunologic dysfunction that ultimately results in one or more opportunistic infections that are associated with profound defects in cellular immunity(1). This immunologic depletion is the result of viral replication in lymphocytes of the CD4+ surface phenotype as well as in cells of monocyte and glial origin.

Agency
National Institute of Health (NIH)
Institute
National Center for Research Resources (NCRR)
Type
General Clinical Research Centers Program (M01)
Project #
3M01RR000865-26S1
Application #
6118432
Study Section
Project Start
1998-12-01
Project End
1999-11-30
Budget Start
1998-10-01
Budget End
1999-09-30
Support Year
26
Fiscal Year
1999
Total Cost
Indirect Cost
Name
University of California Los Angeles
Department
Type
DUNS #
119132785
City
Los Angeles
State
CA
Country
United States
Zip Code
90095
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