Dr. Correa will work with a very experienced administrative person at Vanderbilt University Medical Center. Mrs. Ruby Welch efficiently manages the financial aspects of the POl, including coordination with the Colombian teams at Universidad del Valle and Accion Fiduciaria. GI Administrative personnel will make the travel arrangements for the investigators and the Advisory Committees. Dr. Correa also works closely with Dr. Robertino Mera, M.D., Ph.D. in Biostatistics. Dr. Mera is the main person responsible for the statistical design and analyses of the data generated by all investigators in the three projects. Dr. Mera visited the Colombian component of the Program Project, designed and monitored the data gathering and editing and has been largely responsible for the statistical analysis in the past. Additionally, statisticians from the Vanderbilt Ingram Cancer Center under the direction of Dr. Yu Shyr will advise and participate actively with the local investigators in the statistical aspects of the projects.
The investigators will be working with Vanderbilt personnel to make all the necessary administrative arrangements needed for the conduct ofthe investigation.
|Soutto, Mohammed; Peng, DunFa; Katsha, Ahmed et al. (2015) Activation of ?-catenin signalling by TFF1 loss promotes cell proliferation and gastric tumorigenesis. Gut 64:1028-39|
|Chaturvedi, R; de Sablet, T; Asim, M et al. (2015) Increased Helicobacter pylori-associated gastric cancer risk in the Andean region of Colombia is mediated by spermine oxidase. Oncogene 34:3429-40|
|Krakowiak, M S; Noto, J M; Piazuelo, M B et al. (2015) Matrix metalloproteinase 7 restrains Helicobacter pylori-induced gastric inflammation and premalignant lesions in the stomach by altering macrophage polarization. Oncogene 34:1865-71|
|Wei, Jinxiong; Noto, Jennifer M; Zaika, Elena et al. (2015) Bacterial CagA protein induces degradation of p53 protein in a p14ARF-dependent manner. Gut 64:1040-8|
|Wroblewski, Lydia E; Piazuelo, M Blanca; Chaturvedi, Rupesh et al. (2015) Helicobacter pylori targets cancer-associated apical-junctional constituents in gastroids and gastric epithelial cells. Gut 64:720-30|
|Kodaman, Nuri; Pazos, Alvaro; Schneider, Barbara G et al. (2014) Human and Helicobacter pylori coevolution shapes the risk of gastric disease. Proc Natl Acad Sci U S A 111:1455-60|
|Whary, Mark T; Muthupalani, Sureshkumar; Ge, Zhongming et al. (2014) Helminth co-infection in Helicobacter pylori infected INS-GAS mice attenuates gastric premalignant lesions of epithelial dysplasia and glandular atrophy and preserves colonization resistance of the stomach to lower bowel microbiota. Microbes Infect 16:345-55|
|Chaturvedi, Rupesh; Asim, Mohammad; Piazuelo, M Blanca et al. (2014) Activation of EGFR and ERBB2 by Helicobacter pylori results in survival of gastric epithelial cells with DNA damage. Gastroenterology 146:1739-51.e14|
|Hardbower, Dana M; Peek Jr, Richard M; Wilson, Keith T (2014) At the Bench: Helicobacter pylori, dysregulated host responses, DNA damage, and gastric cancer. J Leukoc Biol 96:201-12|
|Lertpiriyapong, Kvin; Whary, Mark T; Muthupalani, Sureshkumar et al. (2014) Gastric colonisation with a restricted commensal microbiota replicates the promotion of neoplastic lesions by diverse intestinal microbiota in the Helicobacter pylori INS-GAS mouse model of gastric carcinogenesis. Gut 63:54-63|
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