The aims of the Administrative Core are to provide: 1) administrative support for the PPG, conformity to guidelines/policies, requisite reporting, and human resource management;and 2) budget oversight and centralization of shared budgetary items. The Core will provide assistance to the director in the handling of correspondence, forms, competitive and non-competitive renewals and acts as a liaison with the institution, the NIH/NHLBI and other entities. The Core will provide essential secretarial/clerical services to all investigators with regards to the preparation of manuscripts, reviews, articles, letters, travel arrangements, and arrangements for visiting scientists. The Core will make arrangements for regular research meetings of the program scientists and assist in organizing program seminars. It coordinates the advisory committees and provides orientation for Core users. All financial matters for the PPG are handled by the core, including the monitoring and status of service contracts, coordinating repair and service schedules.

Public Health Relevance

The Administrative Core ofthe PPG, monitors conformity to guidelines/policies, provides requisite reporting, human resource management, budget oversight, and centralization of shared budgetary items.

Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Research Program Projects (P01)
Project #
2P01HL028982-31A1
Application #
8460621
Study Section
Heart, Lung, and Blood Initial Review Group (HLBP)
Project Start
Project End
Budget Start
2013-02-15
Budget End
2014-01-31
Support Year
31
Fiscal Year
2013
Total Cost
$226,824
Indirect Cost
$71,995
Name
Henry Ford Health System
Department
Type
DUNS #
073134603
City
Detroit
State
MI
Country
United States
Zip Code
48202
Ramseyer, Vanesa D; Gonzalez-Vicente, Agustin; Carretero, Oscar A et al. (2015) Angiotensin II-induced hypertension blunts thick ascending limb NO production by reducing NO synthase 3 expression and enhancing threonine 495 phosphorylation. Am J Physiol Renal Physiol 308:F149-56
Kassem, Kamal M; Clevenger, Margarette H; Szandzik, David L et al. (2014) PGE2 reduces MMP-14 and increases plasminogen activator inhibitor-1 in cardiac fibroblasts. Prostaglandins Other Lipid Mediat 113-115:62-8
Cabral, Pablo D; Hong, Nancy J; Hye Khan, Md Abdul et al. (2014) Fructose stimulates Na/H exchange activity and sensitizes the proximal tubule to angiotensin II. Hypertension 63:e68-73
Ren, YiLin; D'Ambrosio, Martin A; Garvin, Jeffrey L et al. (2014) Mechanism of impaired afferent arteriole myogenic response in Dahl salt-sensitive rats: role of 20-HETE. Am J Physiol Renal Physiol 307:F533-8
Gonzalez, German E; Rhaleb, Nour-Eddine; Nakagawa, Pablo et al. (2014) N-acetyl-seryl-aspartyl-lysyl-proline reduces cardiac collagen cross-linking and inflammation in angiotensin II-induced hypertensive rats. Clin Sci (Lond) 126:85-94
Xu, Jiang; Sun, Ying; Carretero, Oscar A et al. (2014) Effects of cardiac overexpression of the angiotensin II type 2 receptor on remodeling and dysfunction in mice post-myocardial infarction. Hypertension 63:1251-9
Peng, Hongmei; Xu, Jiang; Yang, Xiao-Ping et al. (2014) Thymosin-?4 prevents cardiac rupture and improves cardiac function in mice with myocardial infarction. Am J Physiol Heart Circ Physiol 307:H741-51
Ren, YiLin; D'Ambrosio, Martin A; Garvin, Jeffrey L et al. (2014) Aldosterone sensitizes connecting tubule glomerular feedback via the aldosterone receptor GPR30. Am J Physiol Renal Physiol 307:F427-34
Ren, Yilin; D'Ambrosio, Martin A; Garvin, Jeffrey L et al. (2013) Prostaglandin E2 mediates connecting tubule glomerular feedback. Hypertension 62:1123-8
Rhaleb, Nour-Eddine; Pokharel, Saraswati; Sharma, Umesh C et al. (2013) N-acetyl-Ser-Asp-Lys-Pro inhibits interleukin-1*-mediated matrix metalloproteinase activation in cardiac fibroblasts. Pflugers Arch 465:1487-95

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