The Administrative Core of this Program Project Grant provides the vital support for all programmatic activities, project investigators, and staff. This Core bridges the activities of the Program with the National Institutes of Health, the Medical College of Wisconsin administrative departments, the Department of Physiology, and the investigators of the Program Project Grant. The staff of this Core is responsible for matters of personnel, purchasing, centralization of service contracts, centralization of biostatistical services, and fiscal management. Core A coordinates annual reports to the National Institutes of Health, programmatic travel by Program Investigators, and visits by invited speakers and/or consultants. Core A also coordinates dissemination of vital information within the Program, organizes scientific meetings and seminars related to the Program, and coordinates the activities of the internal and external advisory committees. The Administrative Core has three Specific Aims:
Aim 1 : Facilitate and coordinate the business requirements associated with the Program Project Grant;
Aim 2 : Facilitate and coordinate communication between the PPG Projects and Cores to maximize scientific progress;
and Aim 3 : Provide statistical support to the Project Investigators.
The Administrative Core is a critical component of this PPG and will support the three scientific projects and the two scientific cores. This Core provides the vital administrative support required to coordinate programmatic activities related to personnel, purchasing, biostatistical services, meetings, and fiscal management.
|Kumar, Vikash; Wollner, Clayton; Kurth, Theresa et al. (2017) Inhibition of Mammalian Target of Rapamycin Complex 1 Attenuates Salt-Induced Hypertension and Kidney Injury in Dahl Salt-Sensitive Rats. Hypertension 70:813-821|
|Mattson, David L; Liang, Mingyu (2017) Hypertension: From GWAS to functional genomics-based precision medicine. Nat Rev Nephrol 13:195-196|
|Abais-Battad, Justine M; Dasinger, John Henry; Fehrenbach, Daniel J et al. (2017) Novel adaptive and innate immunity targets in hypertension. Pharmacol Res 120:109-115|
|Evans, Louise C; Petrova, Galina; Kurth, Theresa et al. (2017) Increased Perfusion Pressure Drives Renal T-Cell Infiltration in the Dahl Salt-Sensitive Rat. Hypertension 70:543-551|
|Hashmat, Shireen; Rudemiller, Nathan; Lund, Hayley et al. (2016) Interleukin-6 inhibition attenuates hypertension and associated renal damage in Dahl salt-sensitive rats. Am J Physiol Renal Physiol 311:F555-61|
|Cowley Jr, Allen W; Yang, Chun; Zheleznova, Nadezhda N et al. (2016) Evidence of the Importance of Nox4 in Production of Hypertension in Dahl Salt-Sensitive Rats. Hypertension 67:440-50|
|Dayton, Alex; Exner, Eric C; Bukowy, John D et al. (2016) Breaking the Cycle: Estrous Variation Does Not Require Increased Sample Size in the Study of Female Rats. Hypertension 68:1139-1144|
|Huang, Baorui; Cheng, Yuan; Usa, Kristie et al. (2016) Renal Tumor Necrosis Factor ? Contributes to Hypertension in Dahl Salt-Sensitive Rats. Sci Rep 6:21960|
|Miller, Bradley; Palygin, Oleg; Rufanova, Victoriya A et al. (2016) p66Shc regulates renal vascular tone in hypertension-induced nephropathy. J Clin Invest 126:2533-46|
|Zheleznova, Nadezhda N; Yang, Chun; Cowley Jr, Allen W (2016) Role of Nox4 and p67phox subunit of Nox2 in ROS production in response to increased tubular flow in the mTAL of Dahl salt-sensitive rats. Am J Physiol Renal Physiol 311:F450-8|
Showing the most recent 10 out of 29 publications