The Administrative Core provides the umbrella for effective operation of the COBRE program. Nearly every activity will be coordinated from this Core. The overall objective of the Administrative Core is to provide an efficient and effective organizational structure in which to ensure good management, integration, and oversight of our COBRE program.
The specific aims of this facility are: 1. To provide skilled personnel for effective fiscal and administrative management of all components of the COBRE program. 2. To provide vision and guidance to each junior investigator in relation to programmatic and career development. 3. To assess the outcomes and success of our tailored team-mentoring plan. 4. To review the scientific accomplishments of the junior investigators and the status of the overall research program with members ofthe Internal and External Advisory Committees and to construct effective means to resolve weaknesses. 5. To provide the interface with NCRR program staff for optimal achievement of program development and long-term goals of this COBRE program.

Public Health Relevance

This Core will provide oversight for the mentoring that is critical to the success of this program. In addition, it will provide critical advice to our investigators on budgets, grant writing and other administrative activities that are critical for our investigators to develop into independently funded researchers.

Agency
National Institute of Health (NIH)
Type
Exploratory Grants (P20)
Project #
5P20GM103629-03
Application #
8663292
Study Section
Special Emphasis Panel (ZRR1)
Project Start
Project End
Budget Start
Budget End
Support Year
3
Fiscal Year
2014
Total Cost
Indirect Cost
Name
Tulane University
Department
Type
DUNS #
City
New Orleans
State
LA
Country
United States
Zip Code
70118
Sweat, Richard S; Sloas, David C; Murfee, Walter L (2014) VEGF-C induces lymphangiogenesis and angiogenesis in the rat mesentery culture model. Microcirculation 21:532-40
Yoshida, Tadashi; Friehs, Ingeborg; Mummidi, Srinivas et al. (2014) Pressure overload induces IL-18 and IL-18R expression, but markedly suppresses IL-18BP expression in a rabbit model. IL-18 potentiates TNF-?-induced cardiomyocyte death. J Mol Cell Cardiol 75:141-51
Yoshida, Tadashi; Huq, Tashfin S; Delafontaine, Patrice (2014) Angiotensin type 2 receptor signaling in satellite cells potentiates skeletal muscle regeneration. J Biol Chem 289:26239-48
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Zsombok, Andrea (2013) Vanilloid receptors--do they have a role in whole body metabolism? Evidence from TRPV1. J Diabetes Complications 27:287-92
Jiang, Yanyan; Gao, Hong; Krantz, Amanda M et al. (2013) Reduced GABAergic inhibition of kidney-related PVN neurons in streptozotocin-treated type 1 diabetic mouse. J Neurophysiol 110:2192-202
Stapor, Peter C; Azimi, Mohammad S; Ahsan, Tabassum et al. (2013) An angiogenesis model for investigating multicellular interactions across intact microvascular networks. Am J Physiol Heart Circ Physiol 304:H235-45

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