The Social Science and Ethics Core (H) provides services which facilitate both domestic and international research efforts in four domains: Research support services, education and training, community engagement, and the development of a HIV/AIDS specific electronic library. This Core supports the conduct of HIV/AIDS behavioral and social science research, the integration of behavioral and social science aspects of HIV/AIDS biomedical research, and the translation of research findings to community. HIV research, particularly intervention, evaluation, and translational research, is interdisciplinary and requires attention to behavioral and contextual issues. Core H provides HIV researchers with resources that enable them to strengthen social science and ethical aspects of their research and/or to augment their ongoing research through the inclusion of components that focus on social science. The Core enhances and facilitates linkages among behavioral/social science, clinical and basic science investigators conducting HIV-related research through the provision of relevant resources and training;facilitates the integration of clinical, biological, and social/behavioral research; and provides leadership to CFAR in the development of meaningful community partnerships that will both enhance research and facilitate the transfer of research findings to the community.

National Institute of Health (NIH)
National Institute of Allergy and Infectious Diseases (NIAID)
Center Core Grants (P30)
Project #
Application #
Study Section
Special Emphasis Panel (ZAI1-JBS-A (J1))
Project Start
Project End
Budget Start
Budget End
Support Year
Fiscal Year
Total Cost
Indirect Cost
Case Western Reserve University
United States
Zip Code
Longenecker, Chris T; Jiang, Ying; Orringer, Carl E et al. (2014) Soluble CD14 is independently associated with coronary calcification and extent of subclinical vascular disease in treated HIV infection. AIDS 28:969-77
Mbonye, Uri; Karn, Jonathan (2014) Transcriptional control of HIV latency: cellular signaling pathways, epigenetics, happenstance and the hope for a cure. Virology 454-455:328-39
Gibson, Richard M; Meyer, Ashley M; Winner, Dane et al. (2014) Sensitive deep-sequencing-based HIV-1 genotyping assay to simultaneously determine susceptibility to protease, reverse transcriptase, integrase, and maturation inhibitors, as well as HIV-1 coreceptor tropism. Antimicrob Agents Chemother 58:2167-85
Chiu, Yen-Ling; Shan, Liang; Huang, Hailiang et al. (2014) Sprouty-2 regulates HIV-specific T cell polyfunctionality. J Clin Invest 124:198-208
Mukherjee, Pranab K; Chandra, Jyotsna; Retuerto, Mauricio et al. (2014) Oral mycobiome analysis of HIV-infected patients: identification of Pichia as an antagonist of opportunistic fungi. PLoS Pathog 10:e1003996
Chandra, Jyotsna; Pearlman, Eric; Ghannoum, Mahmoud A (2014) Animal models to investigate fungal biofilm formation. Methods Mol Biol 1147:141-57
Olvera-GarcĂ­a, Gustavo; Espinosa, Enrique; Sieg, Scott F et al. (2014) Cytomegalovirus-specific responses of CD38? memory T cells are skewed towards IFN-? and dissociated from CD154 in HIV-1 infection. AIDS 28:311-6
Tabler, Caroline O; Lucera, Mark B; Haqqani, Aiman A et al. (2014) CD4+ memory stem cells are infected by HIV-1 in a manner regulated in part by SAMHD1 expression. J Virol 88:4976-86
Zapanta Rinonos, Serendipity; Rai, Urvashi; Vereb, Sydney et al. (2014) Sequential logic of polarity determination during the haploid-to-diploid transition in Saccharomyces cerevisiae. Eukaryot Cell 13:1393-402
Hulgan, Todd; Boger, M Sean; Liao, Diana H et al. (2014) Urinary eicosanoid metabolites in HIV-infected women with central obesity switching to raltegravir: an analysis from the women, integrase, and fat accumulation trial. Mediators Inflamm 2014:803095

Showing the most recent 10 out of 320 publications