The Administrative Core of the UCSF Diabetes and Endocrinology Research Center (DERC) provides leadership, infrastructure, administrative support, and advice and oversight to the other components and members of the DERC. The Center Director (or when absent, the Associate Director) provides day-to-day leadership and interactions with the Directors of the Programs and Cores that constitute the DERC, administrative staff and DERC membership. The Center Director, together with the Directors of the four Research Programs (in Islet Biology, Obesity &Metabolism, Autoimmunity &Inflammation, and Translation) constitute the Executive Committee. The Executive Committee meets quarterly and, when necessary on an ad hoc basis, to set long-range goals in consultation with the Directors of the Pilot &Feasibility and Enrichment Programs and with a Core Coordinating Committee consisting of all four Core Directors and an overall Director of Cores. The Core Coordinating Committee discusses issues and solutions related to Core operations and the provision of Core services to the DERC membership. The Executive Committee also evaluates the external reviews from the Pilot &Feasibility applications and ensures continuing mentorship and advice to Pilot &Feasibility grant recipients. The Executive Committee meets annually with an External Advisory Committee which provides valuable insights into the scientific and operational directions of the DERC. A biannual meeting of the Executive Committee with an Internal Advisory Committee, which consists of institutional leaders with interests in DERC-related research, provides valuable insights about DERC progress in relationship with University goals. The DERC interfaces with its membership, the NIDDK, other NIH Diabetes Centers, and the lay community through this organizational structure and through outreach programs including the Enrichment Program, the Pilot &Feasibility grant program and its web site and other informational materials.
The Objectives of the UCSF DERC are to provide an organizational structure that fosters research collaborations and interactions that accelerate the pace of diabetes research. The Administrative component is charged with providing leadership that responds nimbly to its constituency and that ensures all activities are coordinated towards common goals.
|Kim, Charles C; Nakamura, Mary C; Hsieh, Christine L (2016) Brain trauma elicits non-canonical macrophage activation states. J Neuroinflammation 13:117|
|Lyles, Courtney R; Seligman, Hilary K; Parker, Melissa M et al. (2016) Financial Strain and Medication Adherence among Diabetes Patients in an Integrated Health Care Delivery System: The Diabetes Study of Northern California (DISTANCE). Health Serv Res 51:610-24|
|Stock, Peter G; German, Michael S (2016) A Path to Insulin Independence: "The End of the Beginning". Cell Stem Cell 18:431-3|
|Faleo, Gaetano; Lee, Karim; Nguyen, Vinh et al. (2016) Assessment of Immune Isolation of Allogeneic Mouse Pancreatic Progenitor Cells by a Macroencapsulation Device. Transplantation 100:1211-8|
|Wilson, Camella G; Tran, Jennifer L; Erion, Derek M et al. (2016) Hepatocyte-Specific Disruption of CD36 Attenuates Fatty Liver and Improves Insulin Sensitivity in HFD-Fed Mice. Endocrinology 157:570-85|
|Robblee, Megan M; Kim, Charles C; Porter Abate, Jess et al. (2016) Saturated Fatty Acids Engage an IRE1Î±-Dependent Pathway to Activate the NLRP3 Inflammasome in Myeloid Cells. Cell Rep 14:2611-23|
|Chan, Alice Y; Punwani, Divya; Kadlecek, Theresa A et al. (2016) A novel human autoimmune syndrome caused by combined hypomorphic and activating mutations in ZAP-70. J Exp Med 213:155-65|
|Shah, A D; Kandula, N R; Lin, F et al. (2016) Less favorable body composition and adipokines in South Asians compared with other US ethnic groups: results from the MASALA and MESA studies. Int J Obes (Lond) 40:639-45|
|Ye, Lin; Wang, Jiaming; Tan, Yuting et al. (2016) Genome editing using CRISPR-Cas9 to create the HPFH genotype in HSPCs: An approach for treating sickle cell disease and Î²-thalassemia. Proc Natl Acad Sci U S A 113:10661-5|
|von Moltke, Jakob; Ji, Ming; Liang, Hong-Erh et al. (2016) Tuft-cell-derived IL-25 regulates an intestinal ILC2-epithelial response circuit. Nature 529:221-5|
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