It is a priority of the CEG and the Career Development Program to identify and develop the careers of promising early stage investigators. To accomplish this goal, the overall objective of the career development program (CDP) is to recruit, enrich, encourage, and provide resources and mentoring activities to investigators at the graduate, postgraduate and early faculty levels with a particular emphasis on environmental health science. The CDP has forged new cross-disciplinary partnerships in areas applicable to environmental health science with basic, epidemiological, and/or clinical studies including studies in molecular genetics and environmental epidemiology and environmental toxicology. To accomplish the overall objective, the CDP has developed a highly innovative approach with three specific.
Aim 1 is to attract, recruit and promote opportunities for junior faculty (within three years from initial appointment) towards development of an independent career interfacing between basic, translational or clinical investigations with application to environmental health science. The Next Generation Biomedical Investigator (NGBI) program is the principal mechanism for achieving this aim.
Aim 2 is to identify investigators with less experience (novice investigators) who are still in trainin with minimal experience but have an interest in examining the intricate role of environmental health science in health and disease. Recruits include physicians in their academic fellowship i.e. post residency, who are obtaining an MS or Ph.D, mentored postdoctoral Ph.D. fellows or advanced (third or fourth year) pre-doctoral research students.
This aim i s achieved by supplementing novice investigators'training with research resources, mentoring and career development opportunities through the New Investigator Scholar (NIS) pathway.
Aim 3 is to enrich training/scientific experiences, promote career opportunities and networking experiences, activities integrated with the Administrative and other CEG cores. The objective is to develop NGBIs and NISs and others new to environmental health science by enriching experiences as an investigator and eventually independent scientist. Thus, these aims are achieved through financial awards, mentorship, access to core resources and other career development activities.

Public Health Relevance

Our environmental health center focuses on understanding how genes and environment affect our health, serving the residents, communities of Ohio, the nation and the globe by generating knowledge that is applicable to the general public and policymakers for healthier living.

National Institute of Health (NIH)
National Institute of Environmental Health Sciences (NIEHS)
Center Core Grants (P30)
Project #
Application #
Study Section
Environmental Health Sciences Review Committee (EHS)
Program Officer
Reinlib, Leslie J
Project Start
Project End
Budget Start
Budget End
Support Year
Fiscal Year
Total Cost
Indirect Cost
University of Cincinnati
Public Health & Prev Medicine
Schools of Medicine
United States
Zip Code
Ho, Shuk-Mei; Rao, Rahul; To, Sarah et al. (2017) Bisphenol A and its analogues disrupt centrosome cycle and microtubule dynamics in prostate cancer. Endocr Relat Cancer 24:83-96
Amos-Kroohs, Robyn M; Davenport, Laurie L; Atanasova, Nina et al. (2017) Developmental manganese neurotoxicity in rats: Cognitive deficits in allocentric and egocentric learning and memory. Neurotoxicol Teratol 59:16-26
Ho, Shuk-Mei; Cheong, Ana; Adgent, Margaret A et al. (2017) Environmental factors, epigenetics, and developmental origin of reproductive disorders. Reprod Toxicol 68:85-104
Newman, Nicholas C; Elam, Sarah; Igoe, Carol et al. (2017) A Community-Academic Partnership to Reduce Lead Exposure From an Elevated Roadway Demolition, Cincinnati, Ohio, 2012. Public Health Rep 132:622-626
Khanal, Tilak; Choi, Kwangmin; Leung, Yuet-Kin et al. (2017) Loss of NR2E3 represses AHR by LSD1 reprogramming, is associated with poor prognosis in liver cancer. Sci Rep 7:10662
Lin, Shan; Ptasinska, Anetta; Chen, Xiaoting et al. (2017) A FOXO1-induced oncogenic network defines the AML1-ETO preleukemic program. Blood 130:1213-1222
Vuong, Ann M; Braun, Joseph M; Yolton, Kimberly et al. (2017) Prenatal and postnatal polybrominated diphenyl ether exposure and visual spatial abilities in children. Environ Res 153:83-92
McGuire, Jennifer L; Depasquale, Erica A; Funk, Adam J et al. (2017) Abnormalities of signal transduction networks in chronic schizophrenia. NPJ Schizophr 3:30
Nebert, Daniel W (2017) Aryl hydrocarbon receptor (AHR): ""pioneer member"" of the basic-helix/loop/helix per-Arnt-sim (bHLH/PAS) family of ""sensors"" of foreign and endogenous signals. Prog Lipid Res 67:38-57
Herrick, Robert L; Buckholz, Jeanette; Biro, Frank M et al. (2017) Polyfluoroalkyl substance exposure in the Mid-Ohio River Valley, 1991-2012. Environ Pollut 228:50-60

Showing the most recent 10 out of 943 publications