The Training Core of the UC San Diego Superfund Program supports the interdisciplinary education of junior scientists to become the next generation of environmental researchers in the Environmental Health Sciences. It is a multidisciplinary program at the crossroads of biology, chemistry and environmental sciences which converge in our Superfund program. Our faculty members are affiliated with one or more of several graduate training programs: Chemistry &Biochemistry, Biological Sciences, Biomedical Sciences, and Neurosciences at UCSD or The Scripps Research Institute graduate program, each of which is independently organized and most are multidisciplinary and multi-departmental. Thus, while the supported graduate students may be based in dramatically different scientific disciplines, they share focused interest in environmental health sciences and participate in educational opportunities through meetings, seminar programs, the Community Engagement Core, the Research Translation Core, teaching opportunities, and required classes focused in molecular toxicology and toxicogenomics, ethics, statistics, and environmental health. Training students from a variety of graduate programs in interdisciplinary approaches to environmental sciences is novel and unique. Our program fosters predoctoral training that specifically addresses issues related to environmental health and such programs are critical for the preparation of young investigators to undertake the research so important to improving our environment and providing key information related to toxics in human health. Thus, the interdisciplinary training of Ph.D. graduate students within the extremely rich environment of the UCSD Superfund Program Center, will continue to create a cadre of dedicated, cutting-edge Environmental Health research leaders for the future.

Public Health Relevance

The next generation of environmental health scientists will need to integrate a broader array of skill sets than any other in history. As our environmental challenges become greater, the cadre of scientific professionals required to analyze and solve them will need to be trained in state-of-the-art technologies in a wide variety of fields such as those represented by our Superfund laboratories at UCSD.

Agency
National Institute of Health (NIH)
Institute
National Institute of Environmental Health Sciences (NIEHS)
Type
Hazardous Substances Basic Research Grants Program (NIEHS) (P42)
Project #
5P42ES010337-13
Application #
8659432
Study Section
Special Emphasis Panel ()
Project Start
Project End
Budget Start
2014-04-01
Budget End
2015-03-31
Support Year
13
Fiscal Year
2014
Total Cost
$154,053
Indirect Cost
$38,492
Name
University of California San Diego
Department
Type
DUNS #
804355790
City
La Jolla
State
CA
Country
United States
Zip Code
92093
Fan, Weiwei; Evans, Ronald M (2017) Exercise Mimetics: Impact on Health and Performance. Cell Metab 25:242-247
Chen, Shujuan; Lu, Wenqi; Yueh, Mei-Fei et al. (2017) Intestinal NCoR1, a regulator of epithelial cell maturation, controls neonatal hyperbilirubinemia. Proc Natl Acad Sci U S A 114:E1432-E1440
Lu, Wenqi; Rettenmeier, Eva; Paszek, Miles et al. (2017) Crypt Organoid Culture as an in Vitro Model in Drug Metabolism and Cytotoxicity Studies. Drug Metab Dispos 45:748-754
Reinders, Megan E; Wardi, Gabriel; Bettencourt, Ricki et al. (2017) Increased Risk of Death, in the Hospital and Outside the Intensive Care Unit, for Patients With Cirrhosis After Cardiac Arrest. Clin Gastroenterol Hepatol 15:1808-1810
Liu, Yuanli; Yu, Chuanbai; Cao, Zhixin et al. (2017) A Highly Sensitive Enzymatic Catalysis System for Trace Detection of Arsenic in Water. Chemistry 23:10289-10292
Vollmann, Elisabeth H; Cao, Lizhi; Amatucci, Aldo et al. (2017) Identification of Novel Fibrosis Modifiers by In Vivo siRNA Silencing. Mol Ther Nucleic Acids 7:314-323
He, Nanhai; Fan, Weiwei; Henriquez, Brian et al. (2017) Metabolic control of regulatory T cell (Treg) survival and function by Lkb1. Proc Natl Acad Sci U S A 114:12542-12547
Shalapour, Shabnam; Lin, Xue-Jia; Bastian, Ingmar N et al. (2017) Inflammation-induced IgA+ cells dismantle anti-liver cancer immunity. Nature 551:340-345
Yoda, Emiko; Paszek, Miles; Konopnicki, Camille et al. (2017) Isothiocyanates induce UGT1A1 in humanized UGT1 mice in a CAR dependent fashion that is highly dependent upon oxidative stress. Sci Rep 7:46489
Koyama, Yukinori; Wang, Ping; Liang, Shuang et al. (2017) Mesothelin/mucin 16 signaling in activated portal fibroblasts regulates cholestatic liver fibrosis. J Clin Invest 127:1254-1270

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