The goal of research translation is to ensure that the program's research Is appropriately applied to current environmental and health issues and, in particular, to the effective management of Superfund wastes to minimize and control exposure and risk. The Research Translation Core (RTC) will communicate with environmental, medical, and emergency response professionals, regulating and regulated communities, and the general public, including communities affected by hazardous wastes and thermal treatment of wastes, academia and the new media. The RTC has reached out to a variety of stakeholders since the Program started in July through factsheets, press releases, website publication, presentations at meetings, and articles in various media, and will continue and expand these efforts. For instance, the RTC will publish an electronic newsletter with articles on research and community outreach activities as well as stories on the general state of science and technology regarding the contaminants of concern. The International Congress on Combustion By-Products and their Health Effects, coordinated by Dr. Dellinger, Project Director, is one of the key venues for research communication. The RTC, along with the Administrative Core, is responsible for reporting to the NIEHS SRP formally and informally. We will further develop and maintain relationships with key personnel in agencies such as the U.S. Environmental Protection Agency, the National Institute for Environmental Health Sciences, the Agency for Toxic Substances and Disease Registry, and the Louisiana Department of Environmental Quality (LDEQ). Working with the LSU Office of Intellectual Property, the LSU System Research Technology Foundation, and the New Orleans Biolnnovation Center in technology transfer we will engage the business community by sending research briefs and updates, and by hosting symposia for researchers and entrepreneurs interested in areas ranging from environmental remediation to public health.

Public Health Relevance

The proposed program is concerned with the human health impacts of fine particles and ultrafine particles contaminated with hazardous substances. Effective communication about research findings and their implications is essential in ensuring that the program's research is appropriately applied to the effective management Superfund wastes to minimize and control exposure and risk.

Agency
National Institute of Health (NIH)
Institute
National Institute of Environmental Health Sciences (NIEHS)
Type
Hazardous Substances Basic Research Grants Program (NIEHS) (P42)
Project #
5P42ES013648-05
Application #
8451498
Study Section
Special Emphasis Panel (ZES1-SET-V)
Project Start
Project End
Budget Start
2013-04-01
Budget End
2014-03-31
Support Year
5
Fiscal Year
2013
Total Cost
$100,279
Indirect Cost
$35,882
Name
Louisiana State University A&M Col Baton Rouge
Department
Type
DUNS #
075050765
City
Baton Rouge
State
LA
Country
United States
Zip Code
70803
Hijano, Diego R; Siefker, David T; Shrestha, Bishwas et al. (2018) Type I Interferon Potentiates IgA Immunity to Respiratory Syncytial Virus Infection During Infancy. Sci Rep 8:11034
Haywood, Benjamin J; White, John R; Cook, Robert L (2018) Investigation of an early season river flood pulse: Carbon cycling in a subtropical estuary. Sci Total Environ 635:867-877
Connick, J Patrick; Reed, James R; Backes, Wayne L (2018) Characterization of Interactions Among CYP1A2, CYP2B4, and NADPH-cytochrome P450 Reductase: Identification of Specific Protein Complexes. Drug Metab Dispos 46:197-203
Potter, Phillip M; Guan, Xia; Lomnicki, Slawomir M (2018) Synergy of iron and copper oxides in the catalytic formation of PCDD/Fs from 2-monochlorophenol. Chemosphere 203:96-103
Harmon, Ashlyn C; Hebert, Valeria Y; Cormier, Stephania A et al. (2018) Particulate matter containing environmentally persistent free radicals induces AhR-dependent cytokine and reactive oxygen species production in human bronchial epithelial cells. PLoS One 13:e0205412
Jaligama, Sridhar; Patel, Vivek S; Wang, Pingli et al. (2018) Radical containing combustion derived particulate matter enhance pulmonary Th17 inflammation via the aryl hydrocarbon receptor. Part Fibre Toxicol 15:20
Dugas, Tammy R (2018) Unraveling mechanisms of toxicant-induced oxidative stress in cardiovascular disease. Curr Opin Toxicol 7:1-8
Feld-Cook, Elisabeth E; Bovenkamp-Langlois, Lisa; Lomnicki, Slawo M (2017) Effect of Particulate Matter Mineral Composition on Environmentally Persistent Free Radical (EPFR) Formation. Environ Sci Technol 51:10396-10402
Chuang, Gin C; Xia, Huijing; Mahne, Sarah E et al. (2017) Environmentally Persistent Free Radicals Cause Apoptosis in HL-1 Cardiomyocytes. Cardiovasc Toxicol 17:140-149
Jaligama, Sridhar; Saravia, Jordy; You, Dahui et al. (2017) Regulatory T cells and IL10 suppress pulmonary host defense during early-life exposure to radical containing combustion derived ultrafine particulate matter. Respir Res 18:15

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