The goal of the Community Outreach Core is to work with residents and local environmental leaders facing potential exposure to contaminants from Superfund sites, as well as more diffused and cumulative exposure risks associated with a variety of industrial activities. The Outreach efforts aim to enhance public health and overall community resilience to environmental pollution.
The specific aims address three fundamental dimensions of community resilience: stakeholders'access to scientific information about hazards;social capital building through stronger networks among community groups;and the capacity of residents to develop adaptive strategies when possible to reduce future risk exposure. The team will work with the Louisiana Environmental Action Network (LEAN), a public-interest organization with over 100 affiliated groups, to reach leaders and members of grassroots environmental organizations. Initial efforts (years one and two) will focus on the heavily industrialized North Baton Rouge community, named by the EPA as the highest-ranked parish/county in Louisiana for chronic human health risk and the 24th highest-ranked county in the country (EPA/RSEI 2008). Residents face potential exposure to air-borne pollutants, contaminants in soil and ground water, and even issues concerning fish consumption. The team will identify residents'concerns and risk perceptions and will facilitate systematic interaction between the community stakeholders and researchers within the Center. The information will be presented at workshops with community leaders/residents and will be posted on the LSU Center's website. During the third and fourth years, the team will expand their efforts to include additional grassroots environmental organizations that are members of LEAN. They will conduct systematic evaluations to determine the overall effectiveness of the outreach efforts. These will include pre-and post-tests of workshop participants to gauge changes in knowledge levels concerning exposure routes, risk-mitigation strategies, regulatory guidelines, and opportunities for public participation in decisions affecting local environmental conditions. Tallies of numbers of participating groups will be a direct measure of the extensiveness and network- strengthening effects of the Core.
The fundamental goal of the Center is to gain understanding of integrated pollutant-particle systems and associated EPFR's, and their impacts on the environment and human health. Toward that end, learning more about the concerns and informational needs of residents of highly polluted communities is relevant to Center researchers, and residents will benefit from systematic delivery of information about risks they face.
|Schwingshackl, Andreas; Kimura, Dai; Rovnaghi, Cynthia R et al. (2016) Regulation of inflammatory biomarkers by intravenous methylprednisolone in pediatric ARDS patients: Results from a double-blind, placebo-controlled randomized pilot trial. Cytokine 77:63-71|
|Potter, Phillip M; Dellinger, Barry; Lomnicki, Slawomir M (2016) Contribution of aluminas and aluminosilicates to the formation of PCDD/Fs on fly ashes. Chemosphere 144:2421-6|
|Stephenson, Erin J; Ragauskas, Alyse; Jaligama, Sridhar et al. (2016) Exposure to environmentally persistent free radicals during gestation lowers energy expenditure and impairs skeletal muscle mitochondrial function in adult mice. Am J Physiol Endocrinol Metab 310:E1003-15|
|Paille, Mary; Reams, Margaret; Argote, Jennifer et al. (2016) Influences on Adaptive Planning to Reduce Flood Risks among Parishes in South Louisiana. Water (Basel) 8:|
|Lam, Nina S N; Reams, Margaret; Li, Kenan et al. (2016) Measuring Community Resilience to Coastal Hazards along the Northern Gulf of Mexico. Nat Hazards Rev 17:|
|Deese, Rachel D; LeBlanc, Madeline R; Cook, Robert L (2016) Surfactant toxicity to Artemia Franciscana and the influence of humic acid and chemical composition. Environ Chem 13:507-516|
|You, Dahui; Saravia, Jordy; Siefker, David et al. (2016) Crawling with Virus: Translational Insights from a Neonatal Mouse Model on the Pathogenesis of Respiratory Syncytial Virus in Infants. J Virol 90:2-4|
|Chuang, Gin C; Xia, Huijing; Mahne, Sarah E et al. (2016) Environmentally Persistent Free Radicals Cause Apoptosis in HL-1 Cardiomyocytes. Cardiovasc Toxicol :|
|Nwosu, Ugwumsinachi G; Roy, Amitava; dela Cruz, Albert Leo N et al. (2016) Formation of environmentally persistent free radical (EPFR) in iron(III) cation-exchanged smectite clay. Environ Sci Process Impacts 18:42-50|
|Reed, James R; Backes, Wayne L (2016) The functional effects of physical interactions involving cytochromes P450: putative mechanisms of action and the extent of these effects in biological membranes. Drug Metab Rev 48:453-69|
Showing the most recent 10 out of 81 publications