Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Project (R01)
Project #
5R01AI052291-02
Application #
6765113
Study Section
Special Emphasis Panel (ZRG1-BM-1 (03))
Program Officer
Sizemore, Christine F
Project Start
2003-07-01
Project End
2005-12-31
Budget Start
2004-01-01
Budget End
2004-12-31
Support Year
2
Fiscal Year
2004
Total Cost
$223,200
Indirect Cost
Name
Children's Hospital of Los Angeles
Department
Type
DUNS #
052277936
City
Los Angeles
State
CA
Country
United States
Zip Code
90027
Andini, Nadya; Nash, Kevin A (2011) Expression of tmRNA in mycobacteria is increased by antimicrobial agents that target the ribosome. FEMS Microbiol Lett 322:172-9
Nash, Kevin A; Brown-Elliott, Barbara A; Wallace Jr, Richard J (2009) A novel gene, erm(41), confers inducible macrolide resistance to clinical isolates of Mycobacterium abscessus but is absent from Mycobacterium chelonae. Antimicrob Agents Chemother 53:1367-76
Nash, Kevin A; Andini, Nadya; Zhang, Yansheng et al. (2006) Intrinsic macrolide resistance in rapidly growing mycobacteria. Antimicrob Agents Chemother 50:3476-8
Andini, Nadya; Nash, Kevin A (2006) Intrinsic macrolide resistance of the Mycobacterium tuberculosis complex is inducible. Antimicrob Agents Chemother 50:2560-2
Nash, Kevin A; Zhang, Yansheng; Brown-Elliott, Barbara A et al. (2005) Molecular basis of intrinsic macrolide resistance in clinical isolates of Mycobacterium fortuitum. J Antimicrob Chemother 55:170-7
Nash, Kevin A (2003) Intrinsic macrolide resistance in Mycobacterium smegmatis is conferred by a novel erm gene, erm(38). Antimicrob Agents Chemother 47:3053-60