Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Project (R01)
Project #
5R01AI060561-02
Application #
6805055
Study Section
Special Emphasis Panel (ZRG1-GMA-2 (50))
Program Officer
Koshy, Rajen
Project Start
2003-09-30
Project End
2008-01-31
Budget Start
2004-02-01
Budget End
2005-01-31
Support Year
2
Fiscal Year
2004
Total Cost
$421,250
Indirect Cost
Name
Rockefeller University
Department
Microbiology/Immun/Virology
Type
Other Domestic Higher Education
DUNS #
071037113
City
New York
State
NY
Country
United States
Zip Code
10065
Laidlaw, Stephen M; Marukian, Svetlana; Gilmore, Rachel H et al. (2017) Tumor Necrosis Factor Inhibits Spread of Hepatitis C Virus Among Liver Cells, Independent From Interferons. Gastroenterology 153:566-578.e5
Dustin, Lynn B (2017) Innate and Adaptive Immune Responses in Chronic HCV Infection. Curr Drug Targets 18:826-843
Dustin, L B; Bartolini, B; Capobianchi, M R et al. (2016) Hepatitis C virus: life cycle in cells, infection and host response, and analysis of molecular markers influencing the outcome of infection and response to therapy. Clin Microbiol Infect 22:826-832
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Dustin, Lynn B; Cashman, Siobhán B; Laidlaw, Stephen M (2014) Immune control and failure in HCV infection--tipping the balance. J Leukoc Biol 96:535-48
Cashman, Siobhán B; Marsden, Brian D; Dustin, Lynn B (2014) The Humoral Immune Response to HCV: Understanding is Key to Vaccine Development. Front Immunol 5:550
Charles, Edgar D; Orloff, Michael I M; Nishiuchi, Eiko et al. (2013) Somatic hypermutations confer rheumatoid factor activity in hepatitis C virus-associated mixed cryoglobulinemia. Arthritis Rheum 65:2430-40
Dustin, Lynn B; Charles, Edgar D (2012) Primary, post-primary and non-specific immunoglobulin M responses in HCV infection. Antivir Ther 17:1449-52
Dustin, Lynn B (2012) Too low to measure, infectious nonetheless. Blood 119:6181-2
Stegmann, Kerstin A; Björkström, Niklas K; Ciesek, Sandra et al. (2012) Interferon ?-stimulated natural killer cells from patients with acute hepatitis C virus (HCV) infection recognize HCV-infected and uninfected hepatoma cells via DNAX accessory molecule-1. J Infect Dis 205:1351-62

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