Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS)
Type
Research Project (R01)
Project #
5R01AR040445-07
Application #
2413971
Study Section
Special Emphasis Panel (ZRG5-TMP (01))
Project Start
1990-05-01
Project End
2001-04-30
Budget Start
1997-05-01
Budget End
1998-04-30
Support Year
7
Fiscal Year
1997
Total Cost
Indirect Cost

  Institution

Name
New York State Department of Health
Department
Type
DUNS #
002436061
City
Menands
State
NY
Country
United States
Zip Code
12204

  Publications

LaRocca, Timothy J; Pathak, Priyadarshini; Chiantia, Salvatore et al. (2013) Proving lipid rafts exist: membrane domains in the prokaryote Borrelia burgdorferi have the same properties as eukaryotic lipid rafts. PLoS Pathog 9:e1003353
Crowley, Jameson T; Toledo, Alvaro M; LaRocca, Timothy J et al. (2013) Lipid exchange between Borrelia burgdorferi and host cells. PLoS Pathog 9:e1003109
Toledo, A; Coleman, J L; Kuhlow, C J et al. (2012) The enolase of Borrelia burgdorferi is a plasminogen receptor released in outer membrane vesicles. Infect Immun 80:359-68
Toledo, A; Anda, P; Escudero, R et al. (2010) Phylogenetic analysis of a virulent Borrelia species isolated from patients with relapsing fever. J Clin Microbiol 48:2484-9
LaRocca, Timothy J; Crowley, Jameson T; Cusack, Brian J et al. (2010) Cholesterol lipids of Borrelia burgdorferi form lipid rafts and are required for the bactericidal activity of a complement-independent antibody. Cell Host Microbe 8:331-42
Malkiel, Susan; Kuhlow, Christopher J; Mena, Patricio et al. (2009) The loss and gain of marginal zone and peritoneal B cells is different in response to relapsing fever and Lyme disease Borrelia. J Immunol 182:498-506
Coleman, James L; Katona, Laura I; Kuhlow, Christopher et al. (2009) Evidence that two ATP-dependent (Lon) proteases in Borrelia burgdorferi serve different functions. PLoS Pathog 5:e1000676
Haile, Woldeab B; Coleman, James L; Benach, Jorge L (2006) Reciprocal upregulation of urokinase plasminogen activator and its inhibitor, PAI-2, by Borrelia burgdorferi affects bacterial penetration and host-inflammatory response. Cell Microbiol 8:1349-60
Gebbia, Joseph A; Coleman, James L; Benach, Jorge L (2004) Selective induction of matrix metalloproteinases by Borrelia burgdorferi via toll-like receptor 2 in monocytes. J Infect Dis 189:113-9
Coleman, James L; Benach, Jorge L (2003) The urokinase receptor can be induced by Borrelia burgdorferi through receptors of the innate immune system. Infect Immun 71:5556-64

Showing the most recent 10 out of 23 publications