Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Research Project (R01)
Project #
3R01CA069044-02S2
Application #
2644670
Study Section
Radiation Study Section (RAD)
Project Start
1996-05-15
Project End
1999-04-30
Budget Start
1997-05-01
Budget End
1998-04-30
Support Year
2
Fiscal Year
1997
Total Cost
Indirect Cost
Name
University of California San Francisco
Department
Radiation-Diagnostic/Oncology
Type
Schools of Medicine
DUNS #
073133571
City
San Francisco
State
CA
Country
United States
Zip Code
94143
Bailey, Susan M; Murnane, John P (2006) Telomeres, chromosome instability and cancer. Nucleic Acids Res 34:2408-17
Murnane, John P (2006) Telomeres and chromosome instability. DNA Repair (Amst) 5:1082-92
Volik, Stanislav; Raphael, Benjamin J; Huang, Guiqing et al. (2006) Decoding the fine-scale structure of a breast cancer genome and transcriptome. Genome Res 16:394-404
Sabatier, Laure; Ricoul, Michelle; Pottier, Geraldine et al. (2005) The loss of a single telomere can result in instability of multiple chromosomes in a human tumor cell line. Mol Cancer Res 3:139-50
Murnane, John P; Sabatier, Laure (2004) Chromosome rearrangements resulting from telomere dysfunction and their role in cancer. Bioessays 26:1164-74
Desmaze, C; Pirzio, L M; Blaise, R et al. (2004) Interstitial telomeric repeats are not preferentially involved in radiation-induced chromosome aberrations in human cells. Cytogenet Genome Res 104:123-30
Morales, Jose F; Snow, Elizabeth T; Murnane, John P (2003) Environmental factors affecting transcription of the human L1 retrotransposon. II. Stressors. Mutagenesis 18:151-8
Bai, Yongli; Murnane, John P (2003) Telomere instability in a human tumor cell line expressing a dominant-negative WRN protein. Hum Genet 113:337-47
Lo, Anthony W I; Sprung, Carl N; Fouladi, Bijan et al. (2002) Chromosome instability as a result of double-strand breaks near telomeres in mouse embryonic stem cells. Mol Cell Biol 22:4836-50
Lo, Anthony W I; Sabatier, Laure; Fouladi, Bijan et al. (2002) DNA amplification by breakage/fusion/bridge cycles initiated by spontaneous telomere loss in a human cancer cell line. Neoplasia 4:531-8

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