Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Research Project (R01)
Project #
5R01CA082363-05
Application #
6721109
Study Section
Metabolic Pathology Study Section (MEP)
Program Officer
Mufson, R Allan
Project Start
2000-02-17
Project End
2005-01-31
Budget Start
2004-02-01
Budget End
2005-01-31
Support Year
5
Fiscal Year
2004
Total Cost
$268,474
Indirect Cost
Name
University of Michigan Ann Arbor
Department
Internal Medicine/Medicine
Type
Schools of Medicine
DUNS #
073133571
City
Ann Arbor
State
MI
Country
United States
Zip Code
48109
Wijayatunge, Ranjula; Holmstrom, Sam R; Foley, Samantha B et al. (2018) Deficiency of the Endocytic Protein Hip1 Leads to Decreased Gdpd3 Expression, Low Phosphocholine, and Kypholordosis. Mol Cell Biol 38:
Mgbemena, Victoria E; Signer, Robert A J; Wijayatunge, Ranjula et al. (2017) Distinct Brca1 Mutations Differentially Reduce Hematopoietic Stem Cell Function. Cell Rep 18:947-960
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Foley, Samantha B; Hildenbrand, Zacariah L; Soyombo, Abigail A et al. (2013) Expression of BCR/ABL p210 from a knockin allele enhances bone marrow engraftment without inducing neoplasia. Cell Rep 5:51-60
Ross, Theodora S (2012) Grace Rock's consent. Endocr Relat Cancer 19:C29-31
Ames, Heather M; Bichakjian, Christopher K; Liu, Grace Y et al. (2011) Huntingtin-interacting protein 1: a Merkel cell carcinoma marker that interacts with c-Kit. J Invest Dermatol 131:2113-20
Oravecz-Wilson, Katherine I; Philips, Steven T; Yilmaz, Omer H et al. (2009) Persistence of leukemia-initiating cells in a conditional knockin model of an imatinib-responsive myeloproliferative disorder. Cancer Cell 16:137-48
Graves, Chiron W; Philips, Steven T; Bradley, Sarah V et al. (2008) Use of a cryptic splice site for the expression of huntingtin interacting protein 1 in select normal and neoplastic tissues. Cancer Res 68:1064-73

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