Agency
National Institute of Health (NIH)
Institute
National Institute of Environmental Health Sciences (NIEHS)
Type
Research Project (R01)
Project #
7R01ES005255-03
Application #
3253533
Study Section
Toxicology Subcommittee 2 (TOX)
Project Start
1991-10-17
Project End
1995-06-30
Budget Start
1991-10-17
Budget End
1992-06-30
Support Year
3
Fiscal Year
1991
Total Cost
Indirect Cost
Name
Wayne State University
Department
Type
Schools of Medicine
DUNS #
City
Detroit
State
MI
Country
United States
Zip Code
48202
Bassett, D; Elbon-Copp, C; Otterbein, S et al. (2001) Inflammatory cell availability affects ozone-induced lung damage. J Toxicol Environ Health A 64:547-65
Bassett, D J; Elbon-Copp, C; Ishii, Y et al. (2000) Lung tissue neutrophil content as a determinant of ozone-induced injury. J Toxicol Environ Health A 60:513-30
Gambone, L M; Elbon, C L; Fryer, A D (1994) Ozone-induced loss of neuronal M2 muscarinic receptor function is prevented by cyclophosphamide. J Appl Physiol 77:1492-9
Bassett, D J; Elbon, C L; Ishii, Y et al. (1994) EDU pretreatment decreases polymorphonuclear leukocyte migration into rat lung airways. Toxicol Appl Pharmacol 127:76-82
Schultheis, A H; Bassett, D J (1994) Guinea pig lung inflammatory cell changes following acute ozone exposure. Lung 172:169-81
Choi, A M; Elbon, C L; Bruce, S A et al. (1994) Messenger RNA levels of lung extracellular matrix proteins during ozone exposure. Lung 172:15-30
Leanderson, P; Zackrisson, A L; Tagesson, C et al. (1994) EDU decreases polymorphonuclear leukocyte production of reactive oxygen intermediates. Res Commun Chem Pathol Pharmacol 84:133-41
Schultheis, A H; Bassett, D J; Fryer, A D (1994) Ozone-induced airway hyperresponsiveness and loss of neuronal M2 muscarinic receptor function. J Appl Physiol 76:1088-97
Bassett, D J; Reichenbaugh, S S (1992) Lung mitochondrial function following oxygen exposure and diethyl maleate-induced depletion of glutathione. Toxicol Appl Pharmacol 115:161-7