Ever since its inception in 1978, the Stanford Cancer Biology Program aims to provide the best possible predoctoral and postdoctoral training in cancer research across the nation. One of the great strengths of the training program is its interdepartmental organization that gathers 62 faculty from 23 basic and clinical departments from the schools of Medicine and Humanities and Sciences at Stanford. The central core of our program is intensive and mentored research training. Our trainees conduct cutting-edge research in key areas of Cancer Biology such as cancer stem cells, animal models for cancer, tumor suppressor gene function, and the development of novel therapies to inhibit tumor growth and metastases. Research topics range from using Drosophila to identify genes involved in growth control, to more translational research in screening for small molecules inhibitors that selectively kill tumor cells, to clinical research that applies microarray technology to determine patients'prognostic outcome from tumor gene-expression profiling. Predoctoral trainees build a solid foundation in the discipline of Cancer Biology through required and elective coursework, laboratory research, grant and thesis writing. Postdoctoral trainees also receive rigorous training through research, attending scientific seminars in Cancer Biology, writing fellowship applications and research manuscripts. All trainees are required to present their research at the Annual Asilomar Conference where the entire Stanford Cancer Biology Community gathers to share data and exchange ideas. Trainees are also highly encouraged to attend and present at national and international conferences. All trainees receive formal instructions on biomedical ethics. Trainees'research progress are reviewed annually. In addition, all trainees have the opportunity to meet the invited scientific leaders to discuss about their research, career goals and opportunities. Besides the outstanding faculty mentorship, students are now exposed to the state of the art teaching facilities and research labs that foster extensive research collaborations that arise due to the rapid advancement of cancer research. Finally, with creation of the Stanford Cancer and Stem Cell Institute and the application to become a NCI designated Cancer Center, the Cancer Biology training program will serve an even more prominent role in supporting cancer education and research at Stanford.

Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Institutional National Research Service Award (T32)
Project #
5T32CA009302-35
Application #
8123458
Study Section
Special Emphasis Panel (ZCA1-RTRB-A (M1))
Program Officer
Lim, Susan E
Project Start
1977-09-15
Project End
2013-06-30
Budget Start
2011-07-01
Budget End
2013-06-30
Support Year
35
Fiscal Year
2011
Total Cost
$856,459
Indirect Cost
Name
Stanford University
Department
Radiation-Diagnostic/Oncology
Type
Schools of Medicine
DUNS #
009214214
City
Stanford
State
CA
Country
United States
Zip Code
94305
Kaiser, Alyssa M; Attardi, Laura D (2018) Deconstructing networks of p53-mediated tumor suppression in vivo. Cell Death Differ 25:93-103
Hu, Chi-Kuo; Brunet, Anne (2018) The African turquoise killifish: A research organism to study vertebrate aging and diapause. Aging Cell 17:e12757
Przybyl, Joanna; Chabon, Jacob J; Spans, Lien et al. (2018) Combination Approach for Detecting Different Types of Alterations in Circulating Tumor DNA in Leiomyosarcoma. Clin Cancer Res 24:2688-2699
Lee, Carolyn S; Mah, Angela; Aros, Cody J et al. (2018) Cancer-Associated Long Noncoding RNA SMRT-2 Controls Epidermal Differentiation. J Invest Dermatol 138:1445-1449
Chen, Binbin; Khodadoust, Michael S; Liu, Chih Long et al. (2018) Profiling Tumor Infiltrating Immune Cells with CIBERSORT. Methods Mol Biol 1711:243-259
Tong, Jiyu; Cao, Guangchao; Zhang, Ting et al. (2018) m6A mRNA methylation sustains Treg suppressive functions. Cell Res 28:253-256
Calo, Eliezer; Gu, Bo; Bowen, Margot E et al. (2018) Tissue-selective effects of nucleolar stress and rDNA damage in developmental disorders. Nature 554:112-117
Barkal, Amira A; Weiskopf, Kipp; Kao, Kevin S et al. (2018) Engagement of MHC class I by the inhibitory receptor LILRB1 suppresses macrophages and is a target of cancer immunotherapy. Nat Immunol 19:76-84
Gu, Bo; Swigut, Tomek; Spencley, Andrew et al. (2018) Transcription-coupled changes in nuclear mobility of mammalian cis-regulatory elements. Science 359:1050-1055
Gwinn, Dana M; Lee, Alex G; Briones-Martin-Del-Campo, Marcela et al. (2018) Oncogenic KRAS Regulates Amino Acid Homeostasis and Asparagine Biosynthesis via ATF4 and Alters Sensitivity to L-Asparaginase. Cancer Cell 33:91-107.e6

Showing the most recent 10 out of 420 publications