The Genetics and Molecular Biology Training Program has as its primary goal the education of carefully selected individuals for the research, teaching, and industrial needs of this country. GMB was established by the fusion of two long-standing Princeton training programs: Genetics and Cell and Molecular Biology, and this fusion has been very successful. We receive approximately 290 applications per year, and our success at attracting the best students in the country continues to improve. Currently, this program contains a 48-member faculty who serve as mentors for 148 graduate students, 117 postdoctoral fellows, and 126 undergraduate majors. The participating members include the faculty of the Department of Molecular Biology (35), and 4 faculty from Chemistry, 2 faculty from Chemical Engineering, 2 faculty from Computer Sciences, 1 faculty from Electrical Engineering, 3 faculty from Ecology and Evolutionary Biology, and 1 faculty from Physics. The faculty provides expertise in biological systems ranging from bacteria to humans, and they offer training in the areas of biochemistry, biophysics, cancer, cell biology, computation &modeling, development, evolution, genetics, genomics, microbiology &virology, neuroscience, and structural biology. Training laboratories are located in five different building complexes that are either newly built or recently renovated. Each of these is well-equipped for modern biological research offering state-of-the-art facilities to all. The training program consists of formal course work individually designed to meet the unique intellectual needs of each student, laboratory rotations, teaching experiences, thesis research, and a diverse array of special activities. While formal training is emphasized in year one, and to a significantly lesser extent in year two, we realize that the PhD is a research degree. Accordingly, we spend considerable effort assisting trainees in identifying an appropriate mentor and in monitoring students'subsequent progress in their research labs. The success of our program is best judged by the success of our graduates. We have awarded 264 PhDs and greater than 95% of these individuals remain actively engaged in performing exciting science.

Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Institutional National Research Service Award (T32)
Project #
5T32GM007388-36
Application #
8286241
Study Section
National Institute of General Medical Sciences Initial Review Group (BRT)
Program Officer
Haynes, Susan R
Project Start
1977-07-01
Project End
2013-06-30
Budget Start
2012-07-01
Budget End
2013-06-30
Support Year
36
Fiscal Year
2012
Total Cost
$1,333,765
Indirect Cost
$63,677
Name
Princeton University
Department
Biochemistry
Type
Schools of Arts and Sciences
DUNS #
002484665
City
Princeton
State
NJ
Country
United States
Zip Code
08544
Zheng, Hanqiu; Shen, Minhong; Zha, Yin-Lian et al. (2014) PKD1 phosphorylation-dependent degradation of SNAIL by SCF-FBXO11 regulates epithelial-mesenchymal transition and metastasis. Cancer Cell 26:358-73
McDonald, Karin R; Sabouri, Nasim; Webb, Christopher J et al. (2014) The Pif1 family helicase Pfh1 facilitates telomere replication and has an RPA-dependent role during telomere lengthening. DNA Repair (Amst) 24:80-6
Lin, Shu; Wein, Samuel; Gonzales-Cope, Michelle et al. (2014) Stable-isotope-labeled histone peptide library for histone post-translational modification and variant quantification by mass spectrometry. Mol Cell Proteomics 13:2450-66
Berman, Gordon J; Choi, Daniel M; Bialek, William et al. (2014) Mapping the stereotyped behaviour of freely moving fruit flies. J R Soc Interface 11:
Perez, Lark J; Karagounis, Theodora K; Hurley, Amanda et al. (2014) Highly Potent, Chemically Stable Quorum Sensing Agonists for Vibrio Cholerae. Chem Sci 5:151-155
Chen, Xiao; Bracht, John R; Goldman, Aaron David et al. (2014) The architecture of a scrambled genome reveals massive levels of genomic rearrangement during development. Cell 158:1187-98
Hjeij, Rim; Onoufriadis, Alexandros; Watson, Christopher M et al. (2014) CCDC151 mutations cause primary ciliary dyskinesia by disruption of the outer dynein arm docking complex formation. Am J Hum Genet 95:257-74
Barry, Rachael M; Bitbol, Anne-Florence; Lorestani, Alexander et al. (2014) Large-scale filament formation inhibits the activity of CTP synthetase. Elife 3:e03638
Schoenfeld, Timothy J; Kloth, Alexander D; Hsueh, Brian et al. (2014) Gap junctions in the ventral hippocampal-medial prefrontal pathway are involved in anxiety regulation. J Neurosci 34:15679-88
Sutterlin, Holly A; Zhang, Sisi; Silhavy, Thomas J (2014) Accumulation of phosphatidic acid increases vancomycin resistance in Escherichia coli. J Bacteriol 196:3214-20

Showing the most recent 10 out of 67 publications