The mission of the UCLA-Caltech MSTP is to promote the education of outstanding physician-scientists. To fulfill this mission, our current goals are to 1) recruit exceptionally bright and accomplished students who exhibit an unusual degree of passion for scientific knowledge and a life-long commitment to research and leadership, 2) help guide admitted students toward outstanding training environments that encourage individual thinking and provide students with the tools needed to develop into accomplished physician-scientists, 3) provide a comprehensive support system to meet the trainees'needs and 4) play an increasingly prominent role in guiding the career development of undergraduate students from under-represented ethnic groups and disadvantaged backgrounds. To accomplish these goals as effectively as possible, the UCLA-Caltech MSTP is run by two equal Co-Directors, three Associate Directors, and a strong administrative team, all of whom are deeply committed to the Program. The Program is structured for an average of eight years of study. An integrated, problem-based medical school curriculum is particularly well suited for MSTP students, due to increased time for independent exploration and increased emphasis on research advances that contributed to current knowledge of disease etiology, diagnosis, and treatment. For their Ph.D. research, students choose mentors from a wide array of science and engineering Ph.D. Programs. The MSTP's commitment to excellence was perhaps most apparent when UCLA and Caltech entered into an affiliation agreement fifteen years ago. This affiliation, which provides an opportunity for two students per year to perform their thesis research at Caltech, not only has increased the number of outstanding mentors available to students, but also appears to have increased the Program's visibility and recruitment success. Substantial institutional support from the David Geffen School of Medicine at UCLA and from Caltech has permitted an increase in the size of the MSTP, with 97 students currently enrolled in the program. The MSTP derives great benefit from recent dramatic improvements in physical facilities at both UCLA and Caltech, from the financial health of the universities, and from the recruitment of a large number of outstanding new faculty members to UCLA and Caltech.
The mission of the UCLA-Caltech MSTP is to promote the training of outstanding physician-scientists. The goal is to transform the future of clinical medicine by educating a cadre of doctors who will engage in both clinical care and biomedical research. Such an integration of clinical medicine and scientific research is likely to lead to an improved understanding of human disease and to the discovery of effective new cures for disease.
|Roussotte, Florence F; Jahanshad, Neda; Hibar, Derrek P et al. (2014) A commonly carried genetic variant in the delta opioid receptor gene, OPRD1, is associated with smaller regional brain volumes: replication in elderly and young populations. Hum Brain Mapp 35:1226-36|
|Wilks, Moses Q; Knowles, Scott M; Wu, Anna M et al. (2014) Improved modeling of in vivo kinetics of slowly diffusing radiotracers for tumor imaging. J Nucl Med 55:1539-44|
|Bulterys, Philip L; Kaplan, Jonathan E; Gutman, Julie (2014) Preventing malaria in HIV-infected pregnant women. Clin Infect Dis 58:660-2|
|Knowles, Scott M; Zettlitz, Kirstin A; Tavaré, Richard et al. (2014) Quantitative immunoPET of prostate cancer xenografts with 89Zr- and 124I-labeled anti-PSCA A11 minibody. J Nucl Med 55:452-9|
|Montoya, Dennis; Inkeles, Megan S; Liu, Phillip T et al. (2014) IL-32 is a molecular marker of a host defense network in human tuberculosis. Sci Transl Med 6:250ra114|
|Herting, Megan M; Colby, John B; Sowell, Elizabeth R et al. (2014) White matter connectivity and aerobic fitness in male adolescents. Dev Cogn Neurosci 7:65-75|
|Patterson, M; Gaeta, X; Loo, K et al. (2014) let-7 miRNAs can act through notch to regulate human gliogenesis. Stem Cell Reports 3:758-73|
|Twu, Olivia; Dessí, Daniele; Vu, Anh et al. (2014) Trichomonas vaginalis homolog of macrophage migration inhibitory factor induces prostate cell growth, invasiveness, and inflammatory responses. Proc Natl Acad Sci U S A 111:8179-84|
|Ho, Victoria M; Dallalzadeh, Liane O; Karathanasis, Nestoras et al. (2014) GluA2 mRNA distribution and regulation by miR-124 in hippocampal neurons. Mol Cell Neurosci 61:1-12|
|Kohn, Lisa A; Seet, Christopher S; Scholes, Jessica et al. (2014) Human lymphoid development in the absence of common ?-chain receptor signaling. J Immunol 192:5050-8|
Showing the most recent 10 out of 152 publications