The purpose of CORE A is to provide administrative support to the Program Leaders, to the Principal Investigators of the Projects and Cores, and to the scientific personnel of the Projects and Cores of the HPSG. CORE A will be located in Dr. Chung's laboratory/office complex at the Gastrointestinal Unit of Massachusetts General Hospital. The main goals of CORE A are to manage and oversee the scientific efforts of the Projects and to coordinate scientific and administrative interactions. Thus, the specific tasks of CORE A will be to: a) facilitate interactions between Program Investigators, Scientific Advisors and administrative personnel, b) to plan and coordinate the External Scientific Advisory Group (ESAG) meeting as well as meetings between Program scientists, c) to organize and coordinate travel for scientific advisors, visiting scientists and Program Investigators, d) to assist in the organization of the seminar series and scientific symposium, e) to facilitate resource sharing including assisting in the speedy execution of material transfer agreements (MTA) and f) to assist the Program Directors and Principal Investigators in preparation of progress reports, financial reports and manuscripts for publication. CORE A will also be responsible for the resolution of any potential conflicts.
CORE A is a crucial administrative and organizational structure that will enable the Principal Investigators and personnel of the Projects and Cores of the HPSG to focus on experimental and scientific progress.
|Rowe, Ian A; Tully, Damien C; Armstrong, Matthew J et al. (2016) Effect of scavenger receptor class B type I antagonist ITX5061 in patients with hepatitis C virus infection undergoing liver transplantation. Liver Transpl 22:287-97|
|Fergusson, J R; HÃ¼hn, M H; Swadling, L et al. (2016) CD161(int)CD8+ T cells: a novel population of highly functional, memory CD8+ T cells enriched within the gut. Mucosal Immunol 9:401-13|
|Attanasio, John; Wherry, E John (2016) Costimulatory and Coinhibitory Receptor Pathways in Infectious Disease. Immunity 44:1052-68|
|Llibre, Alba; LÃ³pez-MacÃas, Constantino; Marafioti, Teresa et al. (2016) LLT1 and CD161 Expression in Human Germinal Centers Promotes B Cell Activation and CXCR4 Downregulation. J Immunol 196:2085-94|
|Kurioka, Ayako; Walker, Lucy J; Klenerman, Paul et al. (2016) MAIT cells: new guardians of the liver. Clin Transl Immunology 5:e98|
|van Wilgenburg, Bonnie; Scherwitzl, Iris; Hutchinson, Edward C et al. (2016) MAIT cells are activated during human viral infections. Nat Commun 7:11653|
|Jeffery, Hannah C; van Wilgenburg, Bonnie; Kurioka, Ayako et al. (2016) Biliary epithelium and liver B cells exposed to bacteria activate intrahepatic MAIT cells through MR1. J Hepatol 64:1118-27|
|Kelly, Christabel; Swadling, Leo; Capone, Stefania et al. (2016) Chronic hepatitis C viral infection subverts vaccine-induced T-cell immunity in humans. Hepatology 63:1455-70|
|Chusri, Pattranuch; Kumthip, Kattareeya; Hong, Jian et al. (2016) HCV induces transforming growth factor Î²1 through activation of endoplasmic reticulum stress and the unfolded protein response. Sci Rep 6:22487|
|Swadling, Leo; Halliday, John; Kelly, Christabel et al. (2016) Highly-Immunogenic Virally-Vectored T-cell Vaccines Cannot Overcome Subversion of the T-cell Response by HCV during Chronic Infection. Vaccines (Basel) 4:|
Showing the most recent 10 out of 128 publications