The primary objective of the Administrative Core is to provide the required administrative support to ensure the integrated, coherent and effective scientific operations of the three projects by alleviating Project Leaders from administrative duties. Another important function of the Administrative Core will be to coordinate consulting services to the grant. Specifically, the Core Objectives are: 1) To provide overall scientific and programmatic leadership (e.g., strategic planning, monitoring and evaluation) to the individual Projects in service of the overall scientific theme of this grant. 2) To oversee and coordinate month-to-month activities of the three Projects in order to facilitate reliable exchange of information, collaborative interactions, synergy and cohesiveness of the Projects'research efforts. 3) Preparation and co-ordination of all progress reports to NIH. 4) Preparation and co-ordination of manuscript preparation and submission. 5) Budget projections to assist the Program Director in overseeing the grant. 6) Organization of all aspects of the formal biannual and other informal meetings of the Group, including teleconferencing. 7) Travel arrangements for the Project Leaders to attend the Group meetings. 8) Preparation and distribution of minutes of the Executive Committee meetings. 9) Co-ordination of all consultant activities, including selection and invitation of consultants, and travel arrangements. 10) Facilitate and implement email and telephone consulting activities. 11) Assist with Invention Disclosures and interactions with the offices of Technology Development of UCSD and Scripps.
Through these processes the Administrative Core will assure the synergy and thematic integration of all work conducted under the auspices of this grant, and alleviate the Project Leaders of administrative duties and expenses. As such, the Administrative Core will serve the scientific objectives of this grant.
|Li, Xia; Semenova, Svetlana; D'Souza, Manoranjan S et al. (2014) Involvement of glutamatergic and GABAergic systems in nicotine dependence: Implications for novel pharmacotherapies for smoking cessation. Neuropharmacology 76 Pt B:554-65|
|D'Souza, Manoranjan S; Markou, Athina (2013) The "stop" and "go" of nicotine dependence: role of GABA and glutamate. Cold Spring Harb Perspect Med 3:|
|Chirapu, Srinivas Reddy; Rotter, Charles J; Miller, Emily L et al. (2013) High specificity in response of the sodium-dependent multivitamin transporter to derivatives of pantothenic acid. Curr Top Med Chem 13:837-42|
|Li, Xia; Risbrough, Victoria B; Cates-Gatto, Chelsea et al. (2013) Comparison of the effects of the GABAB receptor positive modulator BHF177 and the GABAB receptor agonist baclofen on anxiety-like behavior, learning, and memory in mice. Neuropharmacology 70:156-67|
|Stoker, Astrid K; Markou, Athina (2013) Unraveling the neurobiology of nicotine dependence using genetically engineered mice. Curr Opin Neurobiol 23:493-9|
|Vlachou, Styliani; Guery, Sebastien; Froestl, Wolfgang et al. (2011) Repeated administration of the GABAB receptor positive modulator BHF177 decreased nicotine self-administration, and acute administration decreased cue-induced reinstatement of nicotine seeking in rats. Psychopharmacology (Berl) 215:117-28|
|D'Souza, Manoranjan S; Markou, Athina (2011) Neuronal mechanisms underlying development of nicotine dependence: implications for novel smoking-cessation treatments. Addict Sci Clin Pract 6:4-16|
|Vlachou, Styliani; Paterson, Neil E; Guery, Sebastien et al. (2011) Both GABA(B) receptor activation and blockade exacerbated anhedonic aspects of nicotine withdrawal in rats. Eur J Pharmacol 655:52-8|
|Wager-Smith, Karen; Markou, Athina (2011) Depression: a repair response to stress-induced neuronal microdamage that can grade into a chronic neuroinflammatory condition? Neurosci Biobehav Rev 35:742-64|