Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Institutional National Research Service Award (T32)
Project #
5T32CA009611-03
Application #
3533716
Study Section
Cancer Institutional Fellowship Review Committee (CT)
Project Start
1988-07-01
Project End
1993-06-30
Budget Start
1990-07-01
Budget End
1991-06-30
Support Year
3
Fiscal Year
1990
Total Cost
Indirect Cost
Name
University of Texas MD Anderson Cancer Center
Department
Type
Other Domestic Higher Education
DUNS #
001910777
City
Houston
State
TX
Country
United States
Zip Code
77030
Roth, J A; Cristiano, R J (1997) Gene therapy for cancer: what have we done and where are we going? J Natl Cancer Inst 89:21-39
Overholt, S M; Liu, T J; Taylor, D L et al. (1997) Head and neck squamous cell growth suppression using adenovirus-p53-FLAG: a potential marker for gene therapy trials. Clin Cancer Res 3:185-91
Roth, J A (1996) Gene replacement strategies for cancer. Isr J Med Sci 32:89-94
Mitchell, M F; Hamada, K; Sastry, K J et al. (1996) Transgene expression in the rhesus cervix mediated by an adenovirus expressing beta-galactosidase. Am J Obstet Gynecol 174:1094-101
Schneider, P M; Casson, A G; Levin, B et al. (1996) Mutations of p53 in Barrett's esophagus and Barrett's cancer: a prospective study of ninety-eight cases. J Thorac Cardiovasc Surg 111:323-31;discussion 331-3
Owen-Schaub, L B; Zhang, W; Cusack, J C et al. (1995) Wild-type human p53 and a temperature-sensitive mutant induce Fas/APO-1 expression. Mol Cell Biol 15:3032-40
Mukhopadhyay, T; Roth, J A (1995) Antisense therapy for cancer. Cancer J Sci Am 1:233-42
Clayman, G L; el-Naggar, A K; Roth, J A et al. (1995) In vivo molecular therapy with p53 adenovirus for microscopic residual head and neck squamous carcinoma. Cancer Res 55:1-6
Fujiwara, T; Grimm, E A; Mukhopadhyay, T et al. (1994) Induction of chemosensitivity in human lung cancer cells in vivo by adenovirus-mediated transfer of the wild-type p53 gene. Cancer Res 54:2287-91
Roth, J A (1994) Modulation of oncogene and tumor-suppressor gene expression: a novel strategy for cancer prevention and treatment. Ann Surg Oncol 1:79-86

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