Agency
National Institute of Health (NIH)
Institute
National Institute on Aging (NIA)
Type
Research Program Projects (P01)
Project #
2P01AG017490-06A1
Application #
7079045
Study Section
Special Emphasis Panel (ZAG1-ZIJ-3 (J2))
Project Start
2006-07-01
Project End
2011-06-30
Budget Start
2006-07-01
Budget End
2007-06-30
Support Year
6
Fiscal Year
2006
Total Cost
$299,260
Indirect Cost
Name
Columbia University (N.Y.)
Department
Type
DUNS #
621889815
City
New York
State
NY
Country
United States
Zip Code
10032
Juranek, Judyta K; Daffu, Gurdip K; Geddis, Matthew S et al. (2016) Soluble RAGE Treatment Delays Progression of Amyotrophic Lateral Sclerosis in SOD1 Mice. Front Cell Neurosci 10:117
Carlson, Emily A; Marquez, Rebecca T; Du, Fang et al. (2015) Overexpression of 17?-hydroxysteroid dehydrogenase type 10 increases pheochromocytoma cell growth and resistance to cell death. BMC Cancer 15:166
Du, Heng; Guo, Lan; Wu, Xiaoping et al. (2014) Cyclophilin D deficiency rescues A?-impaired PKA/CREB signaling and alleviates synaptic degeneration. Biochim Biophys Acta 1842:2517-27
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Juranek, Judyta K; Geddis, Matthew S; Song, Fei et al. (2013) RAGE deficiency improves postinjury sciatic nerve regeneration in type 1 diabetic mice. Diabetes 62:931-43
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Huang, Tianwen; Fang, Fang; Chen, Limin et al. (2012) Ginsenoside Rg1 attenuates oligomeric A?(1-42)-induced mitochondrial dysfunction. Curr Alzheimer Res 9:388-95
Carnevale, Daniela; Mascio, Giada; D'Andrea, Ivana et al. (2012) Hypertension induces brain ?-amyloid accumulation, cognitive impairment, and memory deterioration through activation of receptor for advanced glycation end products in brain vasculature. Hypertension 60:188-97
Yan, Shirley ShiDu; Chen, Doris; Yan, Shiqian et al. (2012) RAGE is a key cellular target for Abeta-induced perturbation in Alzheimer's disease. Front Biosci (Schol Ed) 4:240-50

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