Abstract

The Cancer Prevention and Control Research Program of the Vermont Cancer Center is dedicated to conducting research that will help reduce the burden of cancer through development and application of the behavioral and population sciences to primary and secondary prevention and to care of patients. The wide range of topics covered by this theme requires the skills of many disciplines and support at many levels. The Vermont Cancer Center provides a setting in which essential transdisciplinary linkages are made and provides crucial resources needed to initiate new lines of research. The research of the program falls under three main headings. 1) secondary prevention and psychosocial intervention for breast cancer; 2) behavioral, clinical and population- based studies related to familial cancer risk; and 3) reduction of tobacco- related cancers. Research in recent years has led to important conclusions and design of strategies to change behaviors related to cancer risk, prevention, and surveillance. Program members are actively involved in translation of their research findings into statewide health programs and policies. The program is under the leadership of Brian Flynn, Sc.D., Professor of Family Practice.

  Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Center Core Grants (P30)
Project #
5P30CA022435-19
Application #
6563763
Study Section
Subcommittee G - Education (NCI)
Project Start
2001-12-01
Project End
2002-11-30
Budget Start
1998-10-01
Budget End
1999-09-30
Support Year
19
Fiscal Year
2002
Total Cost
$78,870
Indirect Cost

  Institution

Name
University of Vermont & St Agric College
Department
Type
DUNS #
066811191
City
Burlington
State
VT
Country
United States
Zip Code
05405

  Publications

Kelemen, Linda E; Abbott, Sarah; Qin, Bo et al. (2017) Cigarette smoking and the association with serous ovarian cancer in African American women: African American Cancer Epidemiology Study (AACES). Cancer Causes Control 28:699-708
Long, Patrick M; Tighe, Scott W; Driscoll, Heather E et al. (2015) Acetate supplementation as a means of inducing glioblastoma stem-like cell growth arrest. J Cell Physiol 230:1929-43
Tsen, Andrew R; Long, Patrick M; Driscoll, Heather E et al. (2014) Triacetin-based acetate supplementation as a chemotherapeutic adjuvant therapy in glioma. Int J Cancer 134:1300-10
Wagner, Darcy E; Bonenfant, Nicholas R; Parsons, Charles S et al. (2014) Comparative decellularization and recellularization of normal versus emphysematous human lungs. Biomaterials 35:3281-97
Long, Patrick M; Tighe, Scott W; Driscoll, Heather E et al. (2013) Acetate supplementation induces growth arrest of NG2/PDGFR?-positive oligodendroglioma-derived tumor-initiating cells. PLoS One 8:e80714
Sokocevic, Dino; Bonenfant, Nicholas R; Wagner, Darcy E et al. (2013) The effect of age and emphysematous and fibrotic injury on the re-cellularization of de-cellularized lungs. Biomaterials 34:3256-69
Long, Patrick M; Moffett, John R; Namboodiri, Aryan M A et al. (2013) N-acetylaspartate (NAA) and N-acetylaspartylglutamate (NAAG) promote growth and inhibit differentiation of glioma stem-like cells. J Biol Chem 288:26188-200
Bonenfant, Nicholas R; Sokocevic, Dino; Wagner, Darcy E et al. (2013) The effects of storage and sterilization on de-cellularized and re-cellularized whole lung. Biomaterials 34:3231-45
Murray, Janet M; Messier, Terri; Rivers, Jami et al. (2012) VDJ recombinase-mediated TCR ? locus gene usage and coding joint processing in peripheral T cells during perinatal and pediatric development. J Immunol 189:2356-64
Wallis, John M; Borg, Zachary D; Daly, Amanda B et al. (2012) Comparative assessment of detergent-based protocols for mouse lung de-cellularization and re-cellularization. Tissue Eng Part C Methods 18:420-32

Showing the most recent 10 out of 219 publications