The Cancer Prevention & Control Program consists of 26 Participating Members, representing total peer-reviewed funding for $8.7 Million in annual direct costs ($9.9 Million in total costs). During the last two years, its Members were responsible for a total of 123 cancer-relevant, peer- reviewed publications 33% of which were intra- and inter-programmatic collaborations. This Program is comprised of 4 sub-programs, which are: Tobacco Control; Nutrition; Cancer Epidemiology and Health Education/Community Outreach. The goals of the Program focus on research to promote overall reductions in cancer incidence, mortality and morbidity. Tobacco usage is the most important single cause of cancer in the United States. Consequently members of this sub-program have focused on the behavioral epidemiology of smoking and have made major progress in identifying factors associated with smoking initiation and cessation. The Nutrition sub-program studies the role of diet in cancer onset and progression, and features two large multi-disciplinary intervention studies: one on breast cancer recurrence and the other on recurrence of colonic adenomatous polyps. The health including effects upon cancer incidence, and has developed some of the major hypotheses currently being tested by the women's Health Initiative (UCSD is a site). Health Education and Community Outreach are exceptionally important in diminishing the impact of cancer on the population. This sub-program has developed a number of proven, effective methods for persuading ethnic and cultural minorities and financially disadvantaged sections of the community in San Diego and Imperial counties to participate in cancer screening programs and clinical trials.

Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Center Core Grants (P30)
Project #
5P30CA023100-19
Application #
6599270
Study Section
Subcommittee G - Education (NCI)
Project Start
2002-06-11
Project End
2003-04-30
Budget Start
Budget End
Support Year
19
Fiscal Year
2002
Total Cost
$183,723
Indirect Cost
Name
University of California San Diego
Department
Type
DUNS #
077758407
City
La Jolla
State
CA
Country
United States
Zip Code
92093
Jiang, Qingfei; Jamieson, Catriona (2018) BET'ing on Dual JAK/BET Inhibition as a Therapeutic Strategy for Myeloproliferative Neoplasms. Cancer Cell 33:3-5
Ramirez, Oscar; Aristizabal, Paula; Zaidi, Alia et al. (2018) Implementing a Childhood Cancer Outcomes Surveillance System Within a Population-Based Cancer Registry. J Glob Oncol :1-11
Liu, Liang; Yang, Lin; Yan, Wei et al. (2018) Chemotherapy Induces Breast Cancer Stemness in Association with Dysregulated Monocytosis. Clin Cancer Res 24:2370-2382
Lwin, Thinzar M; Murakami, Takashi; Miyake, Kentaro et al. (2018) Tumor-Specific Labeling of Pancreatic Cancer Using a Humanized Anti-CEA Antibody Conjugated to a Near-Infrared Fluorophore. Ann Surg Oncol 25:1079-1085
Singh, Siddharth; Loomba, Rohit (2018) Role of two-dimensional shear wave elastography in the assessment of chronic liver diseases. Hepatology 67:13-15
Hartman, Sheri J; Nelson, Sandahl H; Myers, Emily et al. (2018) Randomized controlled trial of increasing physical activity on objectively measured and self-reported cognitive functioning among breast cancer survivors: The memory & motion study. Cancer 124:192-202
Hoffmann, Hanne M; Gong, Ping; Tamrazian, Anika et al. (2018) Transcriptional interaction between cFOS and the homeodomain-binding transcription factor VAX1 on the GnRH promoter controls Gnrh1 expression levels in a GnRH neuron maturation specific manner. Mol Cell Endocrinol 461:143-154
Liu, Xuxiang; Cao, Minghui; Palomares, Melanie et al. (2018) Metastatic breast cancer cells overexpress and secrete miR-218 to regulate type I collagen deposition by osteoblasts. Breast Cancer Res 20:127
Huang, Justin K; Carlin, Daniel E; Yu, Michael Ku et al. (2018) Systematic Evaluation of Molecular Networks for Discovery of Disease Genes. Cell Syst 6:484-495.e5
Kalyanaraman, Hema; Schwaerzer, Gerburg; Ramdani, Ghania et al. (2018) Protein Kinase G Activation Reverses Oxidative Stress and Restores Osteoblast Function and Bone Formation in Male Mice With Type 1 Diabetes. Diabetes 67:607-623

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