Abstract

The University of Kansas Cancer Center (KUCC) administration provides critical support to KUCC members in order to facilitate and enhance scientific collaboration and productivity. KUCC is a matrix organization with its members located at the University of Kansas Medieval Center (KUMC), the University of Kansas, Lawrence (KU) and the Stowers Institute for Medical Research (Stowers). The overall objective of the KUCC administrative office is to provide a functional unit that coordinates the entire operation of KUCC, including its shared resources and research programs. This is accomplished by: 1. Providing support for faculty research interactions, including intra- and inter-programmatic and interinstitutional activities;2. Enhancing faculty scientific progress through coordination of KUCC shared resources and grant development and submission support;and 3. Communicating effectively with members about KUCC events and opportunities. KUCC administration is comprised of 10.1 employees who implement the organization and policies that create the Cancer Center infrastructure.

Public Health Relevance

KUCC administration provides support to enhance scientific efforts at the University of Kansas Cancer Center. Administrative functions include financial oversight of the Cancer Center, faculty recruitment support, internal and external communications, grants development support, and research program and shared resource support.

  Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Center Core Grants (P30)
Project #
5P30CA168524-03
Application #
8702108
Study Section
Subcommittee G - Education (NCI)
Project Start
Project End
Budget Start
2014-07-01
Budget End
2015-06-30
Support Year
3
Fiscal Year
2014
Total Cost
$76,416
Indirect Cost

  Institution

Name
University of Kansas
Department
Type
DUNS #
016060860
City
Kansas City
State
KS
Country
United States
Zip Code
66160

  Publications

Li, Linda Xiaoyan; Zhou, Julie Xia; Calvet, James P et al. (2018) Lysine methyltransferase SMYD2 promotes triple negative breast cancer progression. Cell Death Dis 9:326
Peng, Weidan; Furuuchi, Narumi; Aslanukova, Ludmila et al. (2018) Elevated HuR in Pancreas Promotes a Pancreatitis-Like Inflammatory Microenvironment That Facilitates Tumor Development. Mol Cell Biol 38:
Atay, Safinur; Wilkey, Daniel W; Milhem, Mohammed et al. (2018) Insights into the Proteome of Gastrointestinal Stromal Tumors-Derived Exosomes Reveals New Potential Diagnostic Biomarkers. Mol Cell Proteomics 17:495-515
Kasemeier-Kulesa, Jennifer C; Romine, Morgan H; Morrison, Jason A et al. (2018) NGF reprograms metastatic melanoma to a bipotent glial-melanocyte neural crest-like precursor. Biol Open 7:
Del Castillo, Erika; Meier, Richard; Chung, Mei et al. (2018) The Microbiomes of Pancreatic and Duodenum Tissue Overlap and are Highly Subject Specific but Differ between Pancreatic Cancer and Non-Cancer Subjects. Cancer Epidemiol Biomarkers Prev :
Cupertino, Ana Paula; Cartujano-Barrera, Francisco; Perales, Jaime et al. (2018) ""Vive Sin Tabaco… ¡Decídete!"" Feasibility and Acceptability of an e-Health Smoking Cessation Informed Decision-Making Tool Integrated in Primary Healthcare in Mexico. Telemed J E Health :
Neums, Lisa; Suenaga, Seiji; Beyerlein, Peter et al. (2018) VaDiR: an integrated approach to Variant Detection in RNA. Gigascience 7:
Preet, Ranjan; Dixon, Dan A (2018) Mutant KRAS Exosomes Influence the Metabolic State of the Colon Microenvironment. Cell Mol Gastroenterol Hepatol 5:647-648
Tomilin, Viktor; Reif, Gail A; Zaika, Oleg et al. (2018) Deficient transient receptor potential vanilloid type 4 function contributes to compromised [Ca2+]i homeostasis in human autosomal-dominant polycystic kidney disease cells. FASEB J 32:4612-4623
Raman, Archana; Parnell, Stephen C; Zhang, Yan et al. (2018) Periostin overexpression in collecting ducts accelerates renal cyst growth and fibrosis in polycystic kidney disease. Am J Physiol Renal Physiol :

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