This proposal provides central support for research in Mental Retardation at UCLA. The Mental Retardation Research Center (MRRC) at UCLA is a multi-funded, interdisciplinary research and teaching program which has the following objectives: Research into problems of mental retardation and related aspects of human development. Research training, at the pre- and postdoctoral levels, of specialists in a variety of disciplines related to our goals. Studies fall into the following categories: Developmental Biology, Neurobiochemistry, Neurophysiology, Behavioral Studies. An emphasis in indicated on applied research. Collaborative relationships are maintained with other UCLA programs in the Neuropsychiatric Institute, and Brain Research Institute, various departments of the School of Medicine, and other schools at UCLA. This application requests funds for administrative and common research support in Core facilities; Administration, New Program Development, Molecular Biology, Neural Cell Culture, Neurohistology, Microcomputers and Media, Statistics and Electronics and Vivarium.

Agency
National Institute of Health (NIH)
Institute
Eunice Kennedy Shriver National Institute of Child Health & Human Development (NICHD)
Type
Center Core Grants (P30)
Project #
5P30HD004612-22
Application #
3102791
Study Section
Mental Retardation Research Committee (HDMR)
Project Start
1990-08-01
Project End
1995-07-31
Budget Start
1992-08-01
Budget End
1993-07-31
Support Year
22
Fiscal Year
1992
Total Cost
Indirect Cost
Name
University of California Los Angeles
Department
Type
Other Domestic Higher Education
DUNS #
119132785
City
Los Angeles
State
CA
Country
United States
Zip Code
90095
Van, Christina; Condro, Michael C; Lov, Kenny et al. (2018) PACAP/PAC1 Regulation of Inflammation via Catecholaminergic Neurons in a Model of Multiple Sclerosis. J Mol Neurosci :
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Krityakiarana, Warin; Zhao, Paul M; Nguyen, Kevin et al. (2016) Erratum to: Proof-of Concept that an Acute Trophic Factors Intervention After Spinal Cord Injury Provides an Adequate Niche for Neuroprotection, Recruitment of Nestin-Expressing Progenitors and Regeneration. Neurochem Res 41:1844
Abad, Catalina; Jayaram, Bhavaani; Becquet, Laurine et al. (2016) VPAC1 receptor (Vipr1)-deficient mice exhibit ameliorated experimental autoimmune encephalomyelitis, with specific deficits in the effector stage. J Neuroinflammation 13:169
Khankan, Rana R; Griffis, Khris G; Haggerty-Skeans, James R et al. (2016) Olfactory Ensheathing Cell Transplantation after a Complete Spinal Cord Transection Mediates Neuroprotective and Immunomodulatory Mechanisms to Facilitate Regeneration. J Neurosci 36:6269-86

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