, Core A The Stanford Alzheimer's Disease Research Center (ADRC) will serve as a shared resource to facilitate and enhance multidisciplinary and interdisciplinary research in Alzheimer's disease and related disorders; our mission is to specialize in the collection, analysis and dissemination of data sets that is relevant to both Alzheimer's disease and Parkinson's disease research. The Administrative Core (Core A) serves as an administrative structure to direct, facilitate and support the goals of the Stanford ADRC. It assures compliance with NIH policy requirements and provides a forum for planning. It will accomplish its goals through the following specific aims: (1) Coordinate activities of the ADRC Cores, committees, and research projects; (2) Establish advisory committees, which will support Center aims through recommendations, guidance, and critiques; (3) Oversee, monitor, and ensure compliance with reporting procedures, policies, and guidelines of the NIH, institutional review board, university, and other relevant bodies; (4) Stimulate innovative research in Alzheimer's disease and related disorders by existing and new investigators; and (5) Promote research and educational collaborations with other Alzheimer's Disease Centers, universities, health professionals, Alzheimer and Parkinson caregivers, and the wider community.

Agency
National Institute of Health (NIH)
Institute
National Institute on Aging (NIA)
Type
Specialized Center (P50)
Project #
5P50AG047366-04
Application #
9483582
Study Section
Special Emphasis Panel (ZAG1)
Project Start
Project End
Budget Start
2018-05-01
Budget End
2019-04-30
Support Year
4
Fiscal Year
2018
Total Cost
Indirect Cost
Name
Stanford University
Department
Type
DUNS #
009214214
City
Stanford
State
CA
Country
United States
Zip Code
94304
Burke, Shanna L; Maramaldi, Peter; Cadet, Tamara et al. (2018) Decreasing hazards of Alzheimer's disease with the use of antidepressants: mitigating the risk of depression and apolipoprotein E. Int J Geriatr Psychiatry 33:200-211
Kim, Jeehyun; Zhang, Kai; Cai, Weidong et al. (2018) Dopamine-related dissociation of cortical and subcortical brain activations in cognitively unimpaired Parkinson's disease patients OFF and ON medications. Neuropsychologia 119:24-33
Qian, Winnie; Fischer, Corinne E; Schweizer, Tom A et al. (2018) Association Between Psychosis Phenotype and APOE Genotype on the Clinical Profiles of Alzheimer's Disease. Curr Alzheimer Res 15:187-194
Gallagher, Damien; Kiss, Alex; Lanctot, Krista et al. (2018) Depression and Risk of Alzheimer Dementia: A Longitudinal Analysis to Determine Predictors of Increased Risk among Older Adults with Depression. Am J Geriatr Psychiatry 26:819-827
Haaksma, Miriam L; Calderón-Larrañaga, Amaia; Olde Rikkert, Marcel G M et al. (2018) Cognitive and functional progression in Alzheimer disease: A prediction model of latent classes. Int J Geriatr Psychiatry 33:1057-1064
Smith, Alexander K; Periyakoil, Vyjeyanthi S (2018) Should We Bury ""The Good Death""? J Am Geriatr Soc 66:856-858
Ramsey, Christine M; Gnjidic, Danijela; Agogo, George O et al. (2018) Longitudinal patterns of potentially inappropriate medication use following incident dementia diagnosis. Alzheimers Dement (N Y) 4:1-10
Hadjichrysanthou, Christoforos; McRae-McKee, Kevin; Evans, Stephanie et al. (2018) Potential Factors Associated with Cognitive Improvement of Individuals Diagnosed with Mild Cognitive Impairment or Dementia in Longitudinal Studies. J Alzheimers Dis 66:587-600
Hanfelt, John J; Peng, Limin; Goldstein, Felicia C et al. (2018) Latent classes of mild cognitive impairment are associated with clinical outcomes and neuropathology: Analysis of data from the National Alzheimer's Coordinating Center. Neurobiol Dis 117:62-71
Burke, Shanna L; Hu, Tianyan; Fava, Nicole M et al. (2018) Sex differences in the development of mild cognitive impairment and probable Alzheimer's disease as predicted by hippocampal volume or white matter hyperintensities. J Women Aging :1-25

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