Agency
National Institute of Health (NIH)
Institute
National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS)
Type
Specialized Center (P50)
Project #
3P50AR042573-01S1
Application #
3105232
Study Section
Arthritis and Musculoskeletal and Skin Diseases Special Grants Review Committee (AMS)
Project Start
1993-09-30
Project End
1998-08-31
Budget Start
1993-09-30
Budget End
1994-08-31
Support Year
1
Fiscal Year
1994
Total Cost
Indirect Cost
Name
University of North Carolina Chapel Hill
Department
Type
Schools of Medicine
DUNS #
078861598
City
Chapel Hill
State
NC
Country
United States
Zip Code
27599
Weintraub, J P; Cohen, P L (1999) Ectopic expression of B7-1 (CD80) on T lymphocytes in autoimmune lpr and gld mice. Clin Immunol 91:302-9
Caricchio, R; Kovalenko, D; Kaufmann, W K et al. (1999) Apoptosis provoked by the oxidative stress inducer menadione (Vitamin K(3)) is mediated by the Fas/Fas ligand system. Clin Immunol 93:65-74
Caricchio, R; Cohen, P L (1999) Spontaneous and induced apoptosis in systemic lupus erythematosus: multiple assays fail to reveal consistent abnormalities. Cell Immunol 198:54-60
Richards, H B; Satoh, M; Jennette, J C et al. (1999) Disparate T cell requirements of two subsets of lupus-specific autoantibodies in pristane-treated mice. Clin Exp Immunol 115:547-53
Richards, H B; Satoh, M; Shaw, M et al. (1998) Interleukin 6 dependence of anti-DNA antibody production: evidence for two pathways of autoantibody formation in pristane-induced lupus. J Exp Med 188:985-90
Booker, J K; Reap, E A; Cohen, P L (1998) Expression and function of Fas on cells damaged by gamma-irradiation in B6 and B6/lpr mice. J Immunol 161:4536-41
Weintraub, J P; Godfrey, V; Wolthusen, P A et al. (1998) Immunological and pathological consequences of mutations in both Fas and Fas ligand. Cell Immunol 186:8-17
Fecho, K; Bentley, S A; Cohen, P L (1998) Mice deficient in fas ligand (gld) or fas (lpr) show few alterations in granulopoiesis. Cell Immunol 188:19-32
Caricchio, R; Reap, E A; Cohen, P L (1998) Fas/Fas ligand interactions are involved in ultraviolet-B-induced human lymphocyte apoptosis. J Immunol 161:241-51
Gangi-Peterson, L; Peterson, S N; Shapiro, L H et al. (1998) bca: an activation-related B-cell gene. Mol Immunol 35:55-63

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