Agency
National Institute of Health (NIH)
Institute
National Institute of Neurological Disorders and Stroke (NINDS)
Type
Specialized Center (P50)
Project #
5P50NS011920-21
Application #
5215063
Study Section
Project Start
Project End
Budget Start
Budget End
Support Year
21
Fiscal Year
1996
Total Cost
Indirect Cost
Raine, Cedric S (2017) Multiple sclerosis: The resolving lesion revealed. J Neuroimmunol 304:2-6
Raine, Cedric S (2014) Autobiography series: a hitchhiker's road to neuropathology. J Neuropathol Exp Neurol 73:270-81
Brosnan, Celia F; Raine, Cedric S (2013) The astrocyte in multiple sclerosis revisited. Glia 61:453-65
D'Aversa, T G; Eugenin, E A; Lopez, L et al. (2013) Myelin basic protein induces inflammatory mediators from primary human endothelial cells and blood-brain barrier disruption: implications for the pathogenesis of multiple sclerosis. Neuropathol Appl Neurobiol 39:270-83
Lutz, Sarah E; Raine, Cedric S; Brosnan, Celia F (2012) Loss of astrocyte connexins 43 and 30 does not significantly alter susceptibility or severity of acute experimental autoimmune encephalomyelitis in mice. J Neuroimmunol 245:8-14
Gaupp, Stefanie; Arezzo, Joseph; Dutta, Dipankar J et al. (2011) On the occurrence of hypomyelination in a transgenic mouse model: a consequence of the myelin basic protein promoter? J Neuropathol Exp Neurol 70:1138-50
Safavi, Farinaz; Feliberti, Jason P; Raine, Cedric S et al. (2011) Role of ?? T cells in antibody production and recovery from SFV demyelinating disease. J Neuroimmunol 235:18-26
Lutz, Sarah E; Zhao, Yongmei; Gulinello, Maria et al. (2009) Deletion of astrocyte connexins 43 and 30 leads to a dysmyelinating phenotype and hippocampal CA1 vacuolation. J Neurosci 29:7743-52
Zhang, Yueting; Argaw, Azeb Tadesse; Gurfein, Blake T et al. (2009) Notch1 signaling plays a role in regulating precursor differentiation during CNS remyelination. Proc Natl Acad Sci U S A 106:19162-7
Omari, Kakuri M; Lutz, Sarah E; Santambrogio, Laura et al. (2009) Neuroprotection and remyelination after autoimmune demyelination in mice that inducibly overexpress CXCL1. Am J Pathol 174:164-76

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