Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS)
Type
Research Project (R01)
Project #
5R01AR038319-09
Application #
2079270
Study Section
General Medicine A Subcommittee 2 (GMA)
Project Start
1986-07-01
Project End
1999-06-30
Budget Start
1996-07-01
Budget End
1997-06-30
Support Year
9
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
University of Texas Sw Medical Center Dallas
Department
Internal Medicine/Medicine
Type
Schools of Medicine
DUNS #
City
Dallas
State
TX
Country
United States
Zip Code
75390

  Publications

van Duivenvoorde, Leonie M; Dorris, Martha L; Satumtira, Nimman et al. (2012) Relationship between inflammation, bone destruction, and osteoproliferation in the HLA-B27/human ýý2 -microglobulin-transgenic rat model of spondylarthritis. Arthritis Rheum 64:3210-9
Taurog, Joel D; Rival, Claudia; van Duivenvoorde, Leonie M et al. (2012) Autoimmune epididymoorchitis is essential to the pathogenesis of male-specific spondylarthritis in HLA-B27-transgenic rats. Arthritis Rheum 64:2518-28
Taurog, Joel D; Dorris, Martha L; Satumtira, Nimman et al. (2009) Spondylarthritis in HLA-B27/human beta2-microglobulin-transgenic rats is not prevented by lack of CD8. Arthritis Rheum 60:1977-84
Fert, Ingrid; Glatigny, Simon; Poulain, Cecile et al. (2008) Correlation between dendritic cell functional defect and spondylarthritis phenotypes in HLA-B27/HUMAN beta2-microglobulin-transgenic rat lines. Arthritis Rheum 58:3425-9
Tran, Tri M; Dorris, Martha L; Satumtira, Nimman et al. (2006) Additional human beta2-microglobulin curbs HLA-B27 misfolding and promotes arthritis and spondylitis without colitis in male HLA-B27-transgenic rats. Arthritis Rheum 54:1317-27
Turner, Matthew J; Sowders, Dawn P; DeLay, Monica L et al. (2005) HLA-B27 misfolding in transgenic rats is associated with activation of the unfolded protein response. J Immunol 175:2438-48
Tran, Tri Minh; Satumtira, Nimman; Dorris, Martha L et al. (2004) HLA-B27 in transgenic rats forms disulfide-linked heavy chain oligomers and multimers that bind to the chaperone BiP. J Immunol 172:5110-9
May, Ekkehard; Dorris, Martha L; Satumtira, Nimman et al. (2003) CD8 alpha beta T cells are not essential to the pathogenesis of arthritis or colitis in HLA-B27 transgenic rats. J Immunol 170:1099-105
Krug, H E; Taurog, J D (2000) HLA-B27 has no effect on the phenotypic expression of progressive ankylosis in ank/ank mice. J Rheumatol 27:1257-9
Gur, H; Geppert, T D; Wacholtz, M C et al. (1999) The cytoplasmic and the transmembrane domains are not sufficient for class I MHC signal transduction. Cell Immunol 191:105-16

Showing the most recent 10 out of 32 publications