Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
Research Project (R01)
Project #
5R01DK046267-03
Application #
2145439
Study Section
Toxicology Subcommittee 2 (TOX)
Project Start
1994-01-01
Project End
1996-12-31
Budget Start
1996-01-01
Budget End
1996-12-31
Support Year
3
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Adirondack Biomedical Research Institute
Department
Type
DUNS #
City
Lake Placid
State
NY
Country
United States
Zip Code
12946
Ichimura, Takaharu; Asseldonk, Edwin J P V; Humphreys, Benjamin D et al. (2008) Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells. J Clin Invest 118:1657-68
Rennert, Paul D; Ichimura, Takaharu; Sizing, Irene D et al. (2006) T cell, Ig domain, mucin domain-2 gene-deficient mice reveal a novel mechanism for the regulation of Th2 immune responses and airway inflammation. J Immunol 177:4311-21
Asmellash, Senait; Stevens, James L; Ichimura, Takaharu (2005) Modulating the endoplasmic reticulum stress response with trans-4,5-dihydroxy-1,2-dithiane prevents chemically induced renal injury in vivo. Toxicol Sci 88:576-84
Bonventre, Joseph V; Zuk, Anna (2004) Ischemic acute renal failure: an inflammatory disease? Kidney Int 66:480-5
Park, Kwon Moo; Kim, Jee In; Ahn, Youngkeun et al. (2004) Testosterone is responsible for enhanced susceptibility of males to ischemic renal injury. J Biol Chem 279:52282-92
Ichimura, Takaharu; Hung, Cheng Chieh; Yang, Soon Ae et al. (2004) Kidney injury molecule-1: a tissue and urinary biomarker for nephrotoxicant-induced renal injury. Am J Physiol Renal Physiol 286:F552-63
Luyckx, Valerie A; Bonventre, Joseph V (2004) Dose of dialysis in acute renal failure. Semin Dial 17:30-6
Bonventre, Joseph V (2004) Pathophysiology of ischemic acute renal failure. Inflammation, lung-kidney cross-talk, and biomarkers. Contrib Nephrol 144:19-30
Bonventre, Joseph V (2003) Molecular response to cytotoxic injury: role of inflammation, MAP kinases, and endoplasmic reticulum stress response. Semin Nephrol 23:439-48
Andreucci, Michele; Michael, Ashour; Kramers, Cornelis et al. (2003) Renal ischemia/reperfusion and ATP depletion/repletion in LLC-PK(1) cells result in phosphorylation of FKHR and FKHRL1. Kidney Int 64:1189-98

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