Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Research Project (R01)
Project #
2R01GM024891-18A1
Application #
2174346
Study Section
Surgery, Anesthesiology and Trauma Study Section (SAT)
Project Start
1977-07-01
Project End
2000-03-31
Budget Start
1996-04-01
Budget End
1997-03-31
Support Year
18
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Harvard University
Department
Surgery
Type
Schools of Medicine
DUNS #
082359691
City
Boston
State
MA
Country
United States
Zip Code
02115
Kyriakides, C; Austen Jr, W G; Wang, Y et al. (2001) Mast cells mediate complement activation after acid aspiration. Shock 16:21-4
Kyriakides, C; Wang, Y; Austen Jr, W G et al. (2001) Sialyl Lewis(x) hybridized complement receptor type 1 moderates acid aspiration injury. Am J Physiol Lung Cell Mol Physiol 281:L1494-9
Kyriakides, C; Austen Jr, W; Wang, Y et al. (2000) Endothelial selectin blockade attenuates lung permeability of experimental acid aspiration. Surgery 128:327-31
Kyriakides, C; Austen Jr, W G; Wang, Y et al. (2000) Neutrophil mediated remote organ injury after lower torso ischemia and reperfusion is selectin and complement dependent. J Trauma 48:32-8
Williams, J P; Pechet, T T; Weiser, M R et al. (1999) Intestinal reperfusion injury is mediated by IgM and complement. J Appl Physiol 86:938-42
Kyriakides, C; Austen Jr, W; Wang, Y et al. (1999) Membrane attack complex of complement and neutrophils mediate the injury of acid aspiration. J Appl Physiol 87:2357-61
Furman, M I; Benoit, S E; Barnard, M R et al. (1998) Increased platelet reactivity and circulating monocyte-platelet aggregates in patients with stable coronary artery disease. J Am Coll Cardiol 31:352-8
Hastie, L E; Patton, W F; Hechtman, H B et al. (1998) Metabolites of the phospholipase D pathway regulate H2O2-induced filamin redistribution in endothelial cells. J Cell Biochem 68:511-24
Shojaee, N; Patton, W F; Chung-Welch, N et al. (1998) Expression and subcellular distribution of filamin isotypes in endothelial cells and pericytes. Electrophoresis 19:323-32
Peyton, B D; Rohrer, M J; Furman, M I et al. (1998) Patients with venous stasis ulceration have increased monocyte-platelet aggregation. J Vasc Surg 27:1109-15;discussion 1115-6

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