Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Research Project (R01)
Project #
5R01GM032651-13
Application #
2176682
Study Section
Microbial Physiology and Genetics Subcommittee 2 (MBC)
Project Start
1983-07-01
Project End
1998-03-31
Budget Start
1996-04-01
Budget End
1997-03-31
Support Year
13
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Public Health Research Institute
Department
Type
DUNS #
City
Newark
State
NY
Country
United States
Zip Code
Shafikhani, Sasha H; Mandic-Mulec, Ines; Strauch, Mark A et al. (2002) Postexponential regulation of sin operon expression in Bacillus subtilis. J Bacteriol 184:564-71
Rodriguez, G M; Gold, B; Gomez, M et al. (1999) Identification and characterization of two divergently transcribed iron regulated genes in Mycobacterium tuberculosis. Tuber Lung Dis 79:287-98
Dussurget, O; Timm, J; Gomez, M et al. (1999) Transcriptional control of the iron-responsive fxbA gene by the mycobacterial regulator IdeR. J Bacteriol 181:3402-8
Lewis, R J; Brannigan, J A; Offen, W A et al. (1998) An evolutionary link between sporulation and prophage induction in the structure of a repressor:anti-repressor complex. J Mol Biol 283:907-12
Dussurget, O; Smith, I (1998) Interdependence of mycobacterial iron regulation, oxidative-stress response and isoniazid resistance. Trends Microbiol 6:354-8
Smith, I; Dussurget, O; Rodriguez, G M et al. (1998) Extra and intracellular expression of Mycobacterium tuberculosis genes. Tuber Lung Dis 79:91-7
Dussurget, O; Rodriguez, M; Smith, I (1996) An ideR mutant of Mycobacterium smegmatis has derepressed siderophore production and an altered oxidative-stress response. Mol Microbiol 22:535-44
Lewis, R J; Brannigan, J A; Smith, I et al. (1996) Crystallisation of the Bacillus subtilis sporulation inhibitor SinR, complexed with its antagonist, SinI. FEBS Lett 378:98-100
Schmitt, M P; Predich, M; Doukhan, L et al. (1995) Characterization of an iron-dependent regulatory protein (IdeR) of Mycobacterium tuberculosis as a functional homolog of the diphtheria toxin repressor (DtxR) from Corynebacterium diphtheriae. Infect Immun 63:4284-9
Mandic-Mulec, I; Doukhan, L; Smith, I (1995) The Bacillus subtilis SinR protein is a repressor of the key sporulation gene spo0A. J Bacteriol 177:4619-27

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