Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Research Project (R01)
Project #
7R01GM033551-06
Application #
3283436
Study Section
Biochemistry Study Section (BIO)
Project Start
1988-12-01
Project End
1992-03-31
Budget Start
1988-12-01
Budget End
1989-03-31
Support Year
6
Fiscal Year
1988
Total Cost
Indirect Cost
Name
University of California San Francisco
Department
Type
Schools of Medicine
DUNS #
073133571
City
San Francisco
State
CA
Country
United States
Zip Code
94143
Pittet, Jean-Francois; Lee, Hyon; Pespeni, Melissa et al. (2005) Stress-induced inhibition of the NF-kappaB signaling pathway results from the insolubilization of the IkappaB kinase complex following its dissociation from heat shock protein 90. J Immunol 174:384-94
Howard, Marybeth; Fischer, Horst; Roux, Jeremie et al. (2003) Mammalian osmolytes and S-nitrosoglutathione promote Delta F508 cystic fibrosis transmembrane conductance regulator (CFTR) protein maturation and function. J Biol Chem 278:35159-67
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Hansen, William J; Ohh, Michael; Moslehi, Javid et al. (2002) Diverse effects of mutations in exon II of the von Hippel-Lindau (VHL) tumor suppressor gene on the interaction of pVHL with the cytosolic chaperonin and pVHL-dependent ubiquitin ligase activity. Mol Cell Biol 22:1947-60
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Hansen, W J; Cowan, N J; Welch, W J (1999) Prefoldin-nascent chain complexes in the folding of cytoskeletal proteins. J Cell Biol 145:265-77
Nagata, H; Hansen, W J; Freeman, B et al. (1998) Mammalian cytosolic DnaJ homologues affect the hsp70 chaperone-substrate reaction cycle, but do not interact directly with nascent or newly synthesized proteins. Biochemistry 37:6924-38
Brown, C R; Hong-Brown, L Q; Welch, W J (1997) Strategies for correcting the delta F508 CFTR protein-folding defect. J Bioenerg Biomembr 29:491-502

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