Agency
National Institute of Health (NIH)
Institute
Eunice Kennedy Shriver National Institute of Child Health & Human Development (NICHD)
Type
Research Project (R01)
Project #
5R01HD024517-07
Application #
2199207
Study Section
Mammalian Genetics Study Section (MGN)
Project Start
1988-08-01
Project End
1997-12-31
Budget Start
1996-01-01
Budget End
1996-12-31
Support Year
7
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Cincinnati Children's Hospital Medical Center
Department
Type
DUNS #
071284913
City
Cincinnati
State
OH
Country
United States
Zip Code
45229
Bell, Sheila M; Schreiner, Claire M; Wert, Susan E et al. (2008) R-spondin 2 is required for normal laryngeal-tracheal, lung and limb morphogenesis. Development 135:1049-58
Bell, Sheila M; Schreiner, Claire M; Waclaw, Ronald R et al. (2003) Sp8 is crucial for limb outgrowth and neuropore closure. Proc Natl Acad Sci U S A 100:12195-200
Valerius, M Todd; Patterson, Larry T; Feng, Yuxin et al. (2002) Hoxa 11 is upstream of Integrin alpha8 expression in the developing kidney. Proc Natl Acad Sci U S A 99:8090-5
Supp, D M; Brueckner, M; Kuehn, M R et al. (1999) Targeted deletion of the ATP binding domain of left-right dynein confirms its role in specifying development of left-right asymmetries. Development 126:5495-504
Bell, S M; Lan, Y; Jiang, R et al. (1998) Exclusion of Jagged2 as a candidate for the legless gene. Mamm Genome 9:778-9
Bell, S M; Schreiner, C M; Scott, W J (1998) The loss of ventral ectoderm identity correlates with the inability to form an AER in the legless hindlimb bud. Mech Dev 74:41-50
Supp, D M; Witte, D P; Potter, S S et al. (1997) Mutation of an axonemal dynein affects left-right asymmetry in inversus viscerum mice. Nature 389:963-6
Supp, D M; Witte, D P; Branford, W W et al. (1996) Sp4, a member of the Sp1-family of zinc finger transcription factors, is required for normal murine growth, viability, and male fertility. Dev Biol 176:284-99
Scott, W J; Collins, M D; Ernst, A N et al. (1994) Enhanced expression of limb malformations and axial skeleton alterations in legless mutants by transplacental exposure to retinoic acid. Dev Biol 164:277-89
Schreiner, C M; Scott Jr, W J; Supp, D M et al. (1993) Correlation of forelimb malformation asymmetries with visceral organ situs in the transgenic mouse insertional mutation, legless. Dev Biol 158:560-2

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