Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Research Project (R01)
Project #
3R01HL025877-10S1
Application #
3338344
Study Section
Cardiovascular and Renal Study Section (CVB)
Project Start
1991-08-15
Project End
1993-07-31
Budget Start
1991-08-15
Budget End
1992-07-31
Support Year
10
Fiscal Year
1991
Total Cost
Indirect Cost
Name
University of Tennessee Health Science Center
Department
Type
Schools of Medicine
DUNS #
941884009
City
Memphis
State
TN
Country
United States
Zip Code
38163
Bealer, Steven L (2002) Systemic angiotensin II alters intrinsic heart rate through central mechanisms. Brain Res Bull 58:61-5
Bealer, S L (2000) Systemic angiotensin II and volume expansion release norepinephrine in the preoptic recess. Brain Res 864:291-7
Bealer, S L (2000) Central control of cardiac baroreflex responses during peripheral hyperosmolality. Am J Physiol Regul Integr Comp Physiol 278:R1157-63
Bealer, S L (1999) Preoptic recess noradrenergic receptors control modification of baroreflex sensitivity by hypertonicity. Am J Physiol 276:R44-51
Mayne, R G; Armstrong, W E; Crowley, W R et al. (1998) Cytoarchitectonic analysis of Fos-immunoreactivity in brainstem neurones following visceral stimuli in conscious rats. J Neuroendocrinol 10:839-47
Bealer, S L (1997) Acute hypertension increases norepinephrine release in the diagonal band of Broca. Brain Res 745:313-6
Bealer, S L (1997) Preoptic recess alpha-adrenoceptors control cardiovascular responses to hyperosmolality. Am J Physiol 272:R1283-9
Wang, Y X; Crofton, J T; Bealer, S L et al. (1997) Sexual dimorphism in regional blood flow responses to vasopressin in conscious rats. Am J Physiol 273:R1126-31
Cabrera, C L; Bealer, S L; Bohr, D F (1996) Central depressor action of nitric oxide is deficient in genetic hypertension. Am J Hypertens 9:237-41
Bealer, S L (1996) Preoptic recess lesions reduce right atrial pressure responses to volume expansion. J Auton Nerv Syst 60:175-81

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