Agency
National Institute of Health (NIH)
Institute
National Institute of Neurological Disorders and Stroke (NINDS)
Type
Research Project (R01)
Project #
5R01NS025161-09
Application #
2265482
Study Section
Neurology B Subcommittee 2 (NEUB)
Project Start
1987-07-01
Project End
2000-02-29
Budget Start
1996-03-01
Budget End
1997-02-28
Support Year
9
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Columbia University (N.Y.)
Department
Pharmacology
Type
Schools of Medicine
DUNS #
167204994
City
New York
State
NY
Country
United States
Zip Code
10032
Goldberg, D J; Foley, M S; Tang, D et al. (2000) Recruitment of the Arp2/3 complex and mena for the stimulation of actin polymerization in growth cones by nerve growth factor. J Neurosci Res 60:458-67
Tang, D; Goldberg, D J (2000) Bundling of microtubules in the growth cone induced by laminin. Mol Cell Neurosci 15:303-13
Grabham, P W; Foley, M; Umeojiako, A et al. (2000) Nerve growth factor stimulates coupling of beta1 integrin to distinct transport mechanisms in the filopodia of growth cones. J Cell Sci 113 ( Pt 17):3003-12
Grabham, P W; Goldberg, D J (1997) Nerve growth factor stimulates the accumulation of beta1 integrin at the tips of filopodia in the growth cones of sympathetic neurons. J Neurosci 17:5455-65
Goldberg, D J; Wu, D Y (1996) Tyrosine phosphorylation and protrusive structures of the growth cone. Perspect Dev Neurobiol 4:183-92
Wu, D Y; Wang, L C; Mason, C A et al. (1996) Association of beta 1 integrin with phosphotyrosine in growth cone filopodia. J Neurosci 16:1470-8
Goldberg, D J; Wu, D Y (1995) Inhibition of formation of filopodia after axotomy by inhibitors of protein tyrosine kinases. J Neurobiol 27:553-60
Goldberg, D J; Wu, D Y (1994) Regulation of events within the growth cone by extracellular cues: tyrosine phosphorylation. Prog Brain Res 103:75-83
Wu, D Y; Goldberg, D J (1993) Regulated tyrosine phosphorylation at the tips of growth cone filopodia. J Cell Biol 123:653-64
Goldberg, D J; Burmeister, D W (1992) Microtubule-based filopodium-like protrusions form after axotomy. J Neurosci 12:4800-7

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