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Winter, M L; Liehr, J G (1996) Possible mechanism of induction of benign prostatic hyperplasia by estradiol and dihydrotestosterone in dogs. Toxicol Appl Pharmacol 136:211-9
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Zhu, B T; Liehr, J G (1996) Inhibition of catechol O-methyltransferase-catalyzed O-methylation of 2- and 4-hydroxyestradiol by quercetin. Possible role in estradiol-induced tumorigenesis. J Biol Chem 271:1357-63
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Wang, M Y; Liehr, J G (1995) Lipid hydroperoxide-induced endogenous DNA adducts in hamsters: possible mechanism of lipid hydroperoxide-mediated carcinogenesis. Arch Biochem Biophys 316:38-46
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Han, X; Liehr, J G (1995) Microsome-mediated 8-hydroxylation of guanine bases of DNA by steroid estrogens: correlation of DNA damage by free radicals with metabolic activation to quinones. Carcinogenesis 16:2571-4
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Winter, M L; Bosland, M C; Wade, D R et al. (1995) Induction of benign prostatic hyperplasia in intact dogs by near-physiological levels of 5 alpha-dihydrotestosterone and 17 beta-estradiol. Prostate 26:325-33
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Han, X; Liehr, J G; Bosland, M C (1995) Induction of a DNA adduct detectable by 32P-postlabeling in the dorsolateral prostate of NBL/Cr rats treated with estradiol-17 beta and testosterone. Carcinogenesis 16:951-4
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Liehr, J G; Ricci, M J; Jefcoate, C R et al. (1995) 4-Hydroxylation of estradiol by human uterine myometrium and myoma microsomes: implications for the mechanism of uterine tumorigenesis. Proc Natl Acad Sci U S A 92:9220-4
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Wang, M Y; Liehr, J G (1995) Induction by estrogens of lipid peroxidation and lipid peroxide-derived malonaldehyde-DNA adducts in male Syrian hamsters: role of lipid peroxidation in estrogen-induced kidney carcinogenesis. Carcinogenesis 16:1941-5
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Li, D; Wang, M; Liehr, J G et al. (1995) DNA adducts induced by lipids and lipid peroxidation products: possible relationships to I-compounds. Mutat Res 344:117-26
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Moorthy, B; Liehr, J; Randerath, E et al. (1995) Evidence from 32P-postlabeling and the use of pentachlorophenol for a novel metabolic activation pathway of diethylstilbestrol and its dimethyl ether in mouse live: likely alpha-hydroxylation of ethyl group(s) followed by sulfate conjugation. Carcinogenesis 16:2643-8
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