The main purpose of the Biomedical Sciences Research Improvement Program (BISRIP) is to make the Center science departments more effective in training students for research in the biomedical sciences fields and to be more effective in increasing staff productivity in these fields. The basic method of implementation of this objective is the availability and use of centrally-shared equipment under the concept of a Biomolecular-Biopolymer Service Center (BIOSER), which will shortly be housed in the AUC Science complex (upon its completion). BISRIP has the following six broad categories wherein twenty-two projects are supported: a) Developmental biology, b) Biochemistry- Molecular Biology, c) Cancer Research, d) Heart, Lung and Blood Research, e) Biomedical Computer Research, and f) Experimental Psychology. Funds are allocated for: 1) institutional support (through the BIOSER Center); 2) faculty development (through project support); and 3) student development (through creating a favorable research environment). Sponsored by the Atlanta University Center Science Research Institute, the BISRIP Program has involved all of the science departments in the Center. With the increased involvement of the medical school in this grant, it is anticipated that future research will be directed more toward specific disease states and toward more involvement of clinical research activities.

Agency
National Institute of Health (NIH)
Institute
National Center for Research Resources (NCRR)
Type
Minority Biomedical Research Support - MBRS (S06)
Project #
5S06RR008006-18
Application #
3565546
Study Section
General Research Support Program Advisory Committee (GRS)
Project Start
1977-06-01
Project End
1991-02-07
Budget Start
1989-02-17
Budget End
1990-02-07
Support Year
18
Fiscal Year
1989
Total Cost
Indirect Cost
Name
Atlanta University Center
Department
Type
DUNS #
City
Atlanta
State
GA
Country
United States
Zip Code
30310
Urso, P; Wirsiy, Y G; Zhang, W et al. (2008) Alterations in CD4+, CD8+, Vgamma3, Vgammadelta, and/or Valpha betaT-lymphocyte expression in lymphoid tissues of progeny after in utero exposure to benzo(alpha)pyrene. J Immunotoxicol 5:293-306
Sanford, G L; Owens, M A; Odusanya, B M (1993) Differential influence of dexamethasone on the activity and synthesis of beta-galactoside specific lectin (galaptin) during postnatal lung development. Exp Lung Res 19:91-104
Han, G; Mack, D; Hang, J et al. (1992) Evidence for heat-stable liver cytosol substance(s) capable of causing oxidative activation of fructose 1,6-bisphosphatase. Biochem Biophys Res Commun 182:600-8
Trottier, R W; Ogolla, F (1990) In vivo plaque-forming cell suppression by methyl 20 beta-dihydroprednisolonate. Steroids 55:279-82
Sanford, G L; Harris-Hooker, S (1990) Stimulation of vascular cell proliferation by beta-galactoside specific lectins. FASEB J 4:2912-8
Mann, D R; Adams, S R; Gould, K G et al. (1989) Evaluation of the possible direct effects of gonadotrophin-releasing hormone analogues on the monkey (Macaca mulatta) testis. J Reprod Fertil 85:89-95
Paulsen, D F; Solursh, M (1988) Microtiter micromass cultures of limb-bud mesenchymal cells. In Vitro Cell Dev Biol 24:138-47
Leitch, G J; Harris-Hooker, S A; Udezulu, I A (1988) Movement of Entamoeba histolytica trophozoites in rat cecum and colon intact mucus blankets and harvested mucus gels. Am J Trop Med Hyg 39:282-7
Hall Jr, A H; Eanes, R Z; Waymack Jr, P P et al. (1988) Acute effects of a superoxide radical-generating system on DNA double-strand stability in Chinese hamster ovary cells. Determination by a modified fluorometric procedure. Mutat Res 198:161-8
Hein, D W; Trinidad, A; Yerokun, T et al. (1988) Genetic control of acetyl coenzyme A-dependent arylamine N-acetyltransferase, hydrazine N-acetyltransferase, and N-hydroxy-arylamine O-acetyltransferase enzymes in C57BL/6J, A/J, AC57F1, and the rapid and slow acetylator A.B6 and B6.A congenic inbred mous Drug Metab Dispos 16:341-7

Showing the most recent 10 out of 51 publications