The Training Grant supports graduate students and postdoctoral fellows in the Cancer Biology Program at Stanford University. This program is interdisciplinary and is composed of faculty from both the School of Medicine and of Humanities and Sciences. The Principal Investigator on the training grant is also the Director of the program and Chairperson of a faculty Committee on Cancer Biology which reports to the University Committee Graduate Studies and to the Dean of the Medical School. The program provides research training leading to the Ph.D. degree in Cancer Biology, and research training for postdoctoral fellows in areas relevant to the molecular and cellular biology of cancer. Training for predoctoral students involves unit course requirements as well as specific course requirements. The latter include the molecular and cellular biology of cancer, basic biochemistry, advanced molecular biology, cell sciences and the responsible conduct of research. Completion of the Ph.D. requires completion of a qualifying exam, an oral defense, and submission of a written thesis. Trainees supported by the training grant are selected annually by the Committee on Cancer Biology from written applications by qualified candidates and from interviews conducted at Stanford. A total of 16 funded positions for predoctoral trainees are available each year. Funding on this training grant is provided for 4 academic years, after which students are supported by faculty preceptors for the duration of their training. The training grant also provides stipend support for 6 postdoctoral fellows per year. These positions are awarded for 2 years and are selected annually by the Committee on Cancer Biology from a pool of applications. The facilities for training consist primarily of research laboratories under the control of individual faculty preceptors in the Schools of Medicine and Humanities and Sciences, and include all common and core facilities in individual departments represented in these schools. The Cancer Biology Program maintains an office staffed by the Director's Administrator for the program.

Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Institutional National Research Service Award (T32)
Project #
3T32CA009302-30S1
Application #
7279490
Study Section
Subcommittee G - Education (NCI)
Program Officer
Aguila, H Nelson
Project Start
1977-09-15
Project End
2007-06-30
Budget Start
2006-07-25
Budget End
2007-06-30
Support Year
30
Fiscal Year
2006
Total Cost
$43,588
Indirect Cost
Name
Stanford University
Department
Pathology
Type
Schools of Medicine
DUNS #
009214214
City
Stanford
State
CA
Country
United States
Zip Code
94305
Kaiser, Alyssa M; Attardi, Laura D (2018) Deconstructing networks of p53-mediated tumor suppression in vivo. Cell Death Differ 25:93-103
Hu, Chi-Kuo; Brunet, Anne (2018) The African turquoise killifish: A research organism to study vertebrate aging and diapause. Aging Cell 17:e12757
Przybyl, Joanna; Chabon, Jacob J; Spans, Lien et al. (2018) Combination Approach for Detecting Different Types of Alterations in Circulating Tumor DNA in Leiomyosarcoma. Clin Cancer Res 24:2688-2699
Lee, Carolyn S; Mah, Angela; Aros, Cody J et al. (2018) Cancer-Associated Long Noncoding RNA SMRT-2 Controls Epidermal Differentiation. J Invest Dermatol 138:1445-1449
Chen, Binbin; Khodadoust, Michael S; Liu, Chih Long et al. (2018) Profiling Tumor Infiltrating Immune Cells with CIBERSORT. Methods Mol Biol 1711:243-259
Tong, Jiyu; Cao, Guangchao; Zhang, Ting et al. (2018) m6A mRNA methylation sustains Treg suppressive functions. Cell Res 28:253-256
Calo, Eliezer; Gu, Bo; Bowen, Margot E et al. (2018) Tissue-selective effects of nucleolar stress and rDNA damage in developmental disorders. Nature 554:112-117
Barkal, Amira A; Weiskopf, Kipp; Kao, Kevin S et al. (2018) Engagement of MHC class I by the inhibitory receptor LILRB1 suppresses macrophages and is a target of cancer immunotherapy. Nat Immunol 19:76-84
Gu, Bo; Swigut, Tomek; Spencley, Andrew et al. (2018) Transcription-coupled changes in nuclear mobility of mammalian cis-regulatory elements. Science 359:1050-1055
Gwinn, Dana M; Lee, Alex G; Briones-Martin-Del-Campo, Marcela et al. (2018) Oncogenic KRAS Regulates Amino Acid Homeostasis and Asparagine Biosynthesis via ATF4 and Alters Sensitivity to L-Asparaginase. Cancer Cell 33:91-107.e6

Showing the most recent 10 out of 420 publications