(taken from the application):
The specific aim of this competitive renewal of our training program in Burns, Trauma and Critical Care is to develop physicians/scientist who are well grounded in physiology, molecular biology and molecular genetics; in addition this program is designed to teach interested individuals to develop hypothesis based research plans, enabling the trainees to transfer developments in new information from bench research to the clinical arena, leading to the development of novel and innovative therapeutic strategies in trauma and critical care. This training program is devoted to two years research training, and candidates for the training program must have an M.D. or Ph.D. degree from an accredited medical or graduate school, only United States citizens or permanent residents are eligible. A two-year research commitment is required, and M.D. trainees are expected to have completed at least three years of post-doctoral training prior to beginning the fellowship. To achieve an understanding of the cellular and molecular mechanisms underlying organ failure in trauma and sepsis, trainees are placed in laboratories under the direction of scientific preceptor where state-of-the-art scientific methods are available and preceptors are committed to the training of physician/scientists. The academic resources of the University of Texas Southwestern Medical Center and Graduate School as well as the Howard Hughes Medical Institute have produced an exciting cadre of outstanding scientific preceptors for this training program. Individuals selected for participation as potential preceptors are nationally/internationally recognized scientists who have a long-term history of collaboration with investigators in the Department of Surgery and in the Divisions of Pediatric Critical Care. The inclusion of faculty from numerous departments across campus has produced a diverse program, allowing trainees to pursue studies in molecular biology/gene therapeutics, cytokine/mediator biology, neurobiology, tissue engineering/repairs/biomaterials, applied cardiopulmonary and microvascular physiology. The diversity of research areas that revolve around injury and inflammatory responses available to potential trainees should enable candidates to examine pathophysiologic mechanisms of trauma, burns and critical care leading to clinical innovations that will ultimately improve outcome of critically ill patients.
| Hodgman, Erica I; Subramanian, Madhu; Wolf, Steven E et al. (2017) The Effect of Illicit Drug Use on Outcomes Following Burn Injury. J Burn Care Res 38:e89-e94 |
| Song, Juquan; Saeman, Melody R; Baer, Lisa A et al. (2017) Exercise Altered the Skeletal Muscle MicroRNAs and Gene Expression Profiles in Burn Rats With Hindlimb Unloading. J Burn Care Res 38:11-19 |
| Saeman, Melody R; Hodgman, Erica I; Burris, Agnes et al. (2016) Epidemiology and outcomes of pediatric burns over 35 years at Parkland Hospital. Burns 42:202-208 |
| Hodgman, Erica I; Saeman, Melody R; Subramanian, Madhu et al. (2016) The Effect of Burn Center Volume on Mortality in a Pediatric Population: An Analysis of the National Burn Repository. J Burn Care Res 37:32-7 |
| Subramanian, Madhu; Hranjec, Tjasa; Liu, Laindy et al. (2016) A case for less workup in near hanging. J Trauma Acute Care Surg 81:925-930 |
| Saeman, Melody R; DeSpain, Kevin; Liu, Ming-Mei et al. (2016) Severe burn increased skeletal muscle loss in mdx mutant mice. J Surg Res 202:372-9 |
| Saeman, Melody R; Burkhalter, Lorrie S; Blackburn, Timothy J et al. (2015) Radiation exposure and safety practices during pediatric central line placement. J Pediatr Surg 50:992-5 |
| Song, Juquan; Saeman, Melody R; De Libero, Jana et al. (2015) Skeletal Muscle Loss is Associated with TNF Mediated Insufficient Skeletal Myogenic Activation After Burn. Shock 44:479-86 |
| Saeman, Melody R; DeSpain, Kevin; Liu, Ming-Mei et al. (2015) Effects of exercise on soleus in severe burn and muscle disuse atrophy. J Surg Res 198:19-26 |
| Brant, Sarah M; Cobert, Michael L; West, LaShondra M et al. (2014) Characterizing cardiac donation after circulatory death: implications for perfusion preservation. Ann Thorac Surg 98:2107-13; discussion 2113-4 |
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