Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
MARC Undergraduate NRSA Institutional Grants (T34)
Project #
5T34GM008498-10
Application #
6627088
Study Section
Minority Programs Review Committee (MPRC)
Program Officer
Zlotnik, Hinda
Project Start
1994-07-01
Project End
2004-05-31
Budget Start
2003-06-01
Budget End
2004-05-31
Support Year
10
Fiscal Year
2003
Total Cost
$277,228
Indirect Cost
Name
York College
Department
Social Sciences
Type
Schools of Arts and Sciences
DUNS #
620128822
City
New York
State
NY
Country
United States
Zip Code
11451
Arsov, Ivica; Adebayo, Adeola; Kucerova-Levisohn, Martina et al. (2011) A role for autophagic protein beclin 1 early in lymphocyte development. J Immunol 186:2201-9
Juszczak, Laura J; Desamero, Ruel Z B (2009) Extension of the tryptophan chi2,1 dihedral angle-W3 band frequency relationship to a full rotation: correlations and caveats. Biochemistry 48:2777-87
Arsov, I; Li, X; Matthews, G et al. (2008) BAC-mediated transgenic expression of fluorescent autophagic protein Beclin 1 reveals a role for Beclin 1 in lymphocyte development. Cell Death Differ 15:1385-95
Zareen, Neela; Yan, Hua; Hopkinson, Angela et al. (2005) Residues in the conserved His domain of fruit fly tRNase Z that function in catalysis are not involved in substrate recognition or binding. J Mol Biol 350:189-99
Dubrovsky, Edward B; Dubrovskaya, Veronica A; Levinger, Louis et al. (2004) Drosophila RNase Z processes mitochondrial and nuclear pre-tRNA 3' ends in vivo. Nucleic Acids Res 32:255-62
Levinger, Louis; Oestreich, Isabel; Florentz, Catherine et al. (2004) A pathogenesis-associated mutation in human mitochondrial tRNALeu(UUR) leads to reduced 3'-end processing and CCA addition. J Mol Biol 337:535-44
Levinger, Louis; Morl, Mario; Florentz, Catherine (2004) Mitochondrial tRNA 3' end metabolism and human disease. Nucleic Acids Res 32:5430-41
Levinger, Louis; Giege, Richard; Florentz, Catherine (2003) Pathology-related substitutions in human mitochondrial tRNA(Ile) reduce precursor 3' end processing efficiency in vitro. Nucleic Acids Res 31:1904-12
Levinger, L; Jacobs, O; James, M (2001) In vitro 3'-end endonucleolytic processing defect in a human mitochondrial tRNA(Ser(UCN)) precursor with the U7445C substitution, which causes non-syndromic deafness. Nucleic Acids Res 29:4334-40
Mohan, A; Levinger, L (2000) The effects of matrices of paired substitutions in mid-acceptor stem on Drosophila tRNA(His) structure and end-processing. J Mol Biol 303:605-16

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