The NIMH Veterinary Medicine Research Branch (VMRB) has successfully provided support to veterinary medical, husbandry and research support to over thirty-two (32) principal investigators in the NIMH IRP with over eighty-six (86) active animal study protocols. In addition, the Branch has provided training and support to over three-hundred (300) animal users within the Institute. Throughout this fiscal year the NIMH Animal Program Director has also served as Director of the NEI animal care and use program. Both programs were again granted full accreditation by the Association for Assessment and Accreditation of Laboratory Animal Care (AAALAC) International during the summer of FY08. Through its commitment to programs of excellence in veterinary health care, animal husbandry, technical support and program administration the NIMH VMRB has provided program support and facilitation necessary to the mission of the NIMH and NIH. The VMRB continues to play a leading role in the NIH Animal Care and Use Program and as an active participant in the laboratory animal science community both on the local and national level.

Project Start
Project End
Budget Start
Budget End
Support Year
2
Fiscal Year
2010
Total Cost
$1,479,499
Indirect Cost
Name
U.S. National Institute of Mental Health
Department
Type
DUNS #
City
State
Country
Zip Code
Weichbrod, Robert H; Blanks, Stephen; Plemons, Theodore et al. (2015) Untapped Potential: Animal care and use programs adopt alternative training and recruitment strategies for individuals with intellectual and developmental disabilities. Lab Animal Sci Prof 3:12-20
Sztein, Jorge; Vasudevan, Kuzhalini; Raber, James (2010) Refinements in the cryopreservation of mouse ovaries. J Am Assoc Lab Anim Sci 49:420-2
Vasudevan, Kuzhalini; Raber, James; Sztein, Jorge (2010) Fertility comparison between wild type and transgenic mice by in vitro fertilization. Transgenic Res 19:587-94
Zhang, Fan; Tang, Zhongshu; Hou, Xu et al. (2009) VEGF-B is dispensable for blood vessel growth but critical for their survival, and VEGF-B targeting inhibits pathological angiogenesis. Proc Natl Acad Sci U S A 106:6152-7