Programmed Cell Death Induced by Oncoprotein E2-PBX1

Smith, Kevin S.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Cancer Institute (NCI)
• Type: Postdoctoral Individual National Research Service Award (F32)
• Project #: 5F32CA066284-02
• Application #: 2109615
• Study Section: Immunology, Virology, and Pathology Study Section (IVP)
• Project Start: 1996-02-08
• Budget Start: 1996-02-08
• Budget End: 1997-02-07
• Support Year: 2
• Fiscal Year: 1996

Institution

• Name: Stanford University
• Department: Pathology
• Type: Schools of Medicine
• DUNS #: 800771545
• City: Stanford
• State: CA
• Country: United States
• Zip Code: 94305

Publications

Smith, Kevin S; Chanda, Sumit K; Lingbeek, Merel et al. (2003) Bmi-1 regulation of INK4A-ARF is a downstream requirement for transformation of hematopoietic progenitors by E2a-Pbx1. Mol Cell 12:393-400

Smith, K S; Jacobs, Y; Chang, C P et al. (1997) Chimeric oncoprotein E2a-Pbx1 induces apoptosis of hematopoietic cells by a p53-independent mechanism that is suppressed by Bcl-2. Oncogene 14:2917-26