Tcdd Mediated Growth Regulation in Mammary Cells

Davis, John W.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Environmental Health Sciences (NIEHS)
• Type: Postdoctoral Individual National Research Service Award (F32)
• Project #: 5F32ES005895-02
• Application #: 6322324
• Study Section: Alcohol and Toxicology Subcommittee 4 (ALTX)
• Program Officer: Shreffler, Carol K
• Project Start: 2000-11-01
• Project End: 2001-05-31
• Budget Start: 2000-11-01
• Budget End: 2001-05-31
• Support Year: 2
• Fiscal Year: 2001
• Total Cost: $21,756

Institution

• Name: University of New Mexico
• Type: Schools of Pharmacy
• DUNS #: 829868723
• City: Albuquerque
• State: NM
• Country: United States
• Zip Code: 87131

Publications

Davis Jr, John W; Burdick, Andrew D; Lauer, Fredine T et al. (2003) The aryl hydrocarbon receptor antagonist, 3'methoxy-4'nitroflavone, attenuates 2,3,7,8-tetrachlorodibenzo-p-dioxin-dependent regulation of growth factor signaling and apoptosis in the MCF-10A cell line. Toxicol Appl Pharmacol 188:42-9

Davis 2nd, J W; Lauer, F T; Burdick, A D et al. (2001) Prevention of apoptosis by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in the MCF-10A cell line: correlation with increased transforming growth factor alpha production. Cancer Res 61:3314-20

Davis 2nd, J W; Melendez, K; Salas, V M et al. (2000) 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) inhibits growth factor withdrawal-induced apoptosis in the human mammary epithelial cell line, MCF-10A. Carcinogenesis 21:881-6