Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Postdoctoral Individual National Research Service Award (F32)
Project #
5F32HL006968-03
Application #
3049704
Study Section
Clinical Sciences Subcommittee 1 (CLN)
Project Start
1987-01-01
Project End
Budget Start
1987-01-01
Budget End
1987-12-31
Support Year
3
Fiscal Year
1987
Total Cost
Indirect Cost
Name
University of Michigan Ann Arbor
Department
Type
Schools of Medicine
DUNS #
791277940
City
Ann Arbor
State
MI
Country
United States
Zip Code
48109
Bruner, C A; Webb, R C (1990) Increased vascular reactivity to Bay K 8644 in genetic hypertension. Pharmacology 41:24-35
Lee, L; Bruner, C A; Webb, R C (1990) Prostanoids contribute to endothelium-dependent coronary vasodilation in guinea pigs. Blood Vessels 27:341-51
Bruner, C A; Webb, R C (1989) Effect of felodipine on blood pressure and vascular reactivity in stroke-prone spontaneously hypertensive rats. J Hypertens 7:31-5
Bruner, C A; Webb, R C (1988) Adrenal-dependent change in vascular reactivity in stroke-prone spontaneously hypertensive rats. Hypertension 12:388-92
Bruner, C A; Webb, R C (1987) Effect of antihypertensive therapy on a vascular change in genetically hypertensive rats. Clin Exp Hypertens A 9:1745-62
Thompson, L P; Bruner, C A; Lamb, F S et al. (1987) Calcium influx and vascular reactivity in systemic hypertension. Am J Cardiol 59:29A-34A
Bruner, C A; Myers, J H; Sing, C F et al. (1986) Genetic association of hypertension and vascular changes in stroke-prone spontaneously hypertensive rats. Hypertension 8:904-10
Bruner, C A; Myers, J H; Sing, C F et al. (1986) Genetic basis for altered vascular responses to ouabain and potassium-free solution in hypertension. Am J Physiol 251:H1276-82