Pathophysiologic Mechanisms of Lung Injury After Bmt - Kenneth Cooke

Abstract

Funding Agency

Agency: National Institute of Health (NIH)
Institute: National Heart, Lung, and Blood Institute (NHLBI)
Type: Clinical Investigator Award (CIA) (K08)
Project #: 1K08HL003565-01
Application #: 2211812
Study Section: Special Emphasis Panel (ZHL1-CSR-Y (M1))

Project Start: 1996-08-01
Project End: 2001-07-31
Budget Start: 1996-08-01
Budget End: 1997-07-31
Support Year: 1
Fiscal Year: 1996
Total Cost
Indirect Cost

Institution

Name: Dana-Farber Cancer Institute
Department
Type
DUNS #: 149617367

City: Boston
State: MA
Country: United States
Zip Code: 02215

Related projects


NIH 2000 K08 HL	Pathophysiologic Mechanisms of Lung Injury After Bmt Cooke, Kenneth R. / University of Michigan Ann Arbor	$122,310
NIH 1999 K08 HL	Pathophysiologic Mechanisms of Lung Injury After Bmt Cooke, Kenneth R. / University of Michigan Ann Arbor
NIH 1998 K08 HL	Pathophysiologic Mechanisms of Lung Injury After Bmt Cooke, Kenneth R. / Dana-Farber Cancer Institute
NIH 1997 K08 HL	Pathophysiologic Mechanisms of Lung Injury After Bmt Cooke, Kenneth R. / Dana-Farber Cancer Institute
NIH 1996 K08 HL	Pathophysiologic Mechanisms of Lung Injury After Bmt Cooke, Kenneth R. / Dana-Farber Cancer Institute

Publications

Gerbitz, Armin; Ewing, Patricia; Olkiewicz, Krystyna et al. (2005) A role for CD54 (intercellular adhesion molecule-1) in leukocyte recruitment to the lung during the development of experimental idiopathic pneumonia syndrome. Transplantation 79:536-42

Gerbitz, Armin; Nickoloff, Brian J; Olkiewicz, Krystyna et al. (2004) A role for tumor necrosis factor-alpha-mediated endothelial apoptosis in the development of experimental idiopathic pneumonia syndrome. Transplantation 78:494-502

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